Professor | Biwen Peng | Asia Growth Partners

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Name	Biwen Peng
Contact Information	pengbiwen@whu.edu.cn
Language ability	Chinese, English
Province	Hubei

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Name

Biwen Peng

Contact Information

pengbiwen@whu.edu.cn

Language ability

Chinese, English

Province

Hubei

University/Lab Affiliation and Title	Wuhan University
Faculty	Basic Medical Sciences
Academic Background & Achievements	1992-1997: Bachelor of Clinical Medicine, Hubei Medical University 1997-2000: Master of Physiology, Wuhan University 2000-2003: PhD in Pathogenic Biology, Wuhan University 2004-2007: Postdoctoral Researcher in Pharmacology, University of Texas Medical Center
Work Experience	1997-2002: Assistant, Department of Physiology, Wuhan University 2002-2004: Lecturer, Department of Physiology, Wuhan University 2008-2013: Associate Professor, Department of Physiology, Wuhan University 2013-present: Professor, Department of Physiology, Wuhan University
Awards	2007: Third Prize, Science and Technology, People's Government of Fujian Province 2011: Outstanding Young Physiologist Academic Paper Award, Zhang Xijun Fund 2011: Second Prize, Wuhan Natural Science Excellent Academic Paper 2013: Young Scientist Travel Award, Chinese Physiological Society, IUPS (Birmingham, UK) 2020: First-Class Undergraduate Course in Hubei Universities Award for 'Essentials of Human Physiology' 2017-2018: Wuhan University Undergraduate Excellent Teaching Performance Award

University/Lab Affiliation and Title

Wuhan University

Faculty

Basic Medical Sciences

Academic Background & Achievements

1992-1997: Bachelor of Clinical Medicine, Hubei Medical University
1997-2000: Master of Physiology, Wuhan University
2000-2003: PhD in Pathogenic Biology, Wuhan University
2004-2007: Postdoctoral Researcher in Pharmacology, University of Texas Medical Center

Work Experience

1997-2002: Assistant, Department of Physiology, Wuhan University
2002-2004: Lecturer, Department of Physiology, Wuhan University
2008-2013: Associate Professor, Department of Physiology, Wuhan University
2013-present: Professor, Department of Physiology, Wuhan University

Awards

2007: Third Prize, Science and Technology, People's Government of Fujian Province
2011: Outstanding Young Physiologist Academic Paper Award, Zhang Xijun Fund
2011: Second Prize, Wuhan Natural Science Excellent Academic Paper
2013: Young Scientist Travel Award, Chinese Physiological Society, IUPS (Birmingham, UK)
2020: First-Class Undergraduate Course in Hubei Universities Award for 'Essentials of Human Physiology'
2017-2018: Wuhan University Undergraduate Excellent Teaching Performance Award

Areas of Focus	Neuroimmune mechanisms of paroxysmal neurological diseases Effects of glial cells on synaptic activity and mechanisms, especially the role of microglia in synapse formation and neuronal activity Mechanisms of active substance release by microglia, providing insights into mechanisms of neonatal epilepsy, hypoxic-ischemic and thermal stress injuries
Publications	Complement C3 Aggravates Post-epileptic Neuronal Injury Via Activation of TRPV1, Peng BW et al., 2021 Silencing of circIgf1r plays a protective role in neuronal injury via regulating astrocyte polarization during epilepsy, Peng BW et al., 2021 NFAT5 and HIF-1α Coordinate to Regulate NKCC1 Expression in Hippocampal Neurons After Hypoxia-Ischemia, Peng BW et al., 2019 TRPV1 mediates astrocyte activation and interleukin-1β release induced by hypoxic ischemia (HI), Peng BW et al., 2019 TRPV1 translocated to astrocytic membrane to promote migration and inflammatory infiltration thus promotes epilepsy after hypoxic ischemia in immature brain, Peng BW et al., 2019 Activation of TRPV1 Contributes to Recurrent Febrile Seizures via Inhibiting the Microglial M2 Phenotype in the Immature Brain, Peng BW et al., 2019 Vitexin reduces epilepsy after hypoxic ischemia in the neonatal brain via inhibition of NKCC1, Peng BW et al., 2018 Modulation of neuroinflammation: Role and therapeutic potential of TRPV1 in the neuro-immune axis, Peng BW et al., 2017

Areas of Focus

Neuroimmune mechanisms of paroxysmal neurological diseases
Effects of glial cells on synaptic activity and mechanisms, especially the role of microglia in synapse formation and neuronal activity
Mechanisms of active substance release by microglia, providing insights into mechanisms of neonatal epilepsy, hypoxic-ischemic and thermal stress injuries

Publications

Complement C3 Aggravates Post-epileptic Neuronal Injury Via Activation of TRPV1, Peng BW et al., 2021
Silencing of circIgf1r plays a protective role in neuronal injury via regulating astrocyte polarization during epilepsy, Peng BW et al., 2021
NFAT5 and HIF-1α Coordinate to Regulate NKCC1 Expression in Hippocampal Neurons After Hypoxia-Ischemia, Peng BW et al., 2019
TRPV1 mediates astrocyte activation and interleukin-1β release induced by hypoxic ischemia (HI), Peng BW et al., 2019
TRPV1 translocated to astrocytic membrane to promote migration and inflammatory infiltration thus promotes epilepsy after hypoxic ischemia in immature brain, Peng BW et al., 2019
Activation of TRPV1 Contributes to Recurrent Febrile Seizures via Inhibiting the Microglial M2 Phenotype in the Immature Brain, Peng BW et al., 2019
Vitexin reduces epilepsy after hypoxic ischemia in the neonatal brain via inhibition of NKCC1, Peng BW et al., 2018
Modulation of neuroinflammation: Role and therapeutic potential of TRPV1 in the neuro-immune axis, Peng BW et al., 2017

Neuroimmune Paroxysmal Diseases Glial Cells Synaptic Activity Microglia Synapse Formation Neuronal Activity Active Substance Release Neonatal Epilepsy Hypoxic-Ischemic Injuries

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