1993.4-1997.3 PhD: Department of Cardiology, University of Tokyo
1979.9-1984.8 Bachelor's: Department of Medicine, Peking University
National Outstanding Youth Science Fund, National Natural Science Foundation of China, 2006
Shanghai Outstanding Academic Leader, Shanghai Science and Technology Commission, 2005
2004.9-present - Fudan University, Zhongshan Hospital - Professor
2005.6-present - Fudan University, Zhongshan Hospital/Biomedical Research Institute - Professor (dual appointment)
2000.4-2004.8 - University of Tokyo Medical School Cardiology and Chiba University Medical School Cardiology - Researcher
1993.4-1997.3 - University of Tokyo Medical School Cardiology - Postdoctoral Fellow
1984.9-1993.3 - Qingdao Medical College Affiliated Hospital Cardiology - Attending Physician
Shanghai Outstanding Overseas Chinese Person (2015): Shanghai Government
Huaxia Medical Third Prize (2014): Health Promotion Association
Chinese Medical Association Science and Technology Third Prize (2014): Chinese Medical Association
Shanghai Science and Technology Progress First Prize (2014): Shanghai Association for Science and Technology
Ministry of Education Science and Technology Progress Second Prize (2014): Ministry of Education
Shanghai Science and Technology Elite (2013): Shanghai Government
Shanghai Medical Science and Technology Second Prize (2012): Shanghai Medical Association
Research
Cardiovascular System Internal Medicine Research Hypertensive Myocardial Hypertrophy and Heart Failure Pathogenesis Angiotensin Function Research Gene and Cell Therapy for Ischemic Heart Disease
Mechanical stresses induce paracrine β-2 microglobulin from cardiomyocytes to activate cardiac fibroblasts through epidermal growth factor receptor., Li Y, Zhang X, Li L, Wang X, Chen Z, Wang X, Wang Y, Kang L, Ye Y, Jia J, Zhang G, Yang C, Yuan J, Zhou J, Ge J, Gong H, Zou Y., 2018
Heat-shock transcription factor 1 is critically involved in the ischaemia-induced cardiac hypertrophy via JAK2/STAT3 pathway., Yuan L, Qiu L, Ye Y, Wu J, Wang S, Wang X, Zhou N, Zou Y., 2018
MicroRNA-378 suppresses myocardial fibrosis through a paracrine mechanism at the early stage of cardiac hypertrophy following mechanical stress., Yuan J, Liu H, Gao W, Zhang L, Ye Y, Yuan L, Ding Z, Wu J, Kang L, Zhang X, Wang X, Zhang G, Gong H, Sun A, Yang X, Chen R, Cui Z, Ge J, Zou Y., 2018
HSF1 deficiency accelerates the transition from pressure overload-induced cardiac hypertrophy to heart failure through endothelial miR-195a-3p-mediated impairment of cardiac angiogenesis., Wang S, Wu J, You J, Shi H, Xue X, Huang J, Xu L, Jiang G, Yuan L, Gong X, Luo H, Ge J, Cui Z, Zou Y., 2018
Cardiomyocyte-Restricted Low Density Lipoprotein Receptor-Related Protein 6 (LRP6) Deletion Leads to Lethal Dilated Cardiomyopathy Partly Through Drp1 Signaling., Chen Z, Li Y, Wang Y, Qian J, Ma H, Wang X, Jiang G, Liu M, An Y, Ma L, Kang L, Jia J, Yang C, Zhang G, Chen Y, Gao W, Fu M, Huang Z, Tang H, Zhu Y, Ge J, Gong H, Zou Y., 2018
Differential cardiac hypertrophy and signaling pathways in pressure versus volume overload., You J, Wu J, Zhang Q, Ye Y, Wang S, Huang J, Liu H, Wang X, Zhang W, Bu L, Li J, Lin L, Ge J, Zou Y., 2018
Opposing Roles of Wnt Inhibitors IGFBP-4 and Dkk1 in Cardiac Ischemia by Differential Targeting of LRP5/6 and β-catenin., Wo D, Peng J, Ren DN, Qiu L, Chen J, Zhu Y, Yan Y, Yan H, Wu J, Ma E, Zhong TP, Chen Y, Liu Z, Liu S, Ao L, Liu Z, Jiang C, Peng J, Zou Y, Qian Q, Zhu W., 2016
Exosomes derived from mature dendritic cells increase endothelial inflammation and atherosclerosis via membrane TNF-α mediated NF-κB pathway., Gao W, Liu H, Yuan J, Wu C, Huang D, Ma Y, Zhu J, Ma L, Guo J, Shi H, Zou Y, Ge J., 2016
Advanced glycation end products impair the functions of saphenous vein but not thoracic artery smooth muscle cells through RAGE/MAPK signalling pathway in diabetes., Sun Y, Kang L, Li J, Liu H, Wang Y, Wang C, Zou Y., 2016
The effects of different angiotensin II type 1 receptor blockers on the regulation of the ACE-AngII-AT1 and ACE2-Ang(1-7)-Mas axes in pressure overload-induced cardiac remodeling in male mice., Wang X, Ye Y, Gong H, Wu J, Yuan J, Wang S, Yin P, Ding Z, Kang L, Jiang Q, Zhang W, Li Y, Ge J, Zou Y., 2016
Mechanical Stress Regulates Endothelial Progenitor Cell Angiogenesis Through VEGF Receptor Endocytosis., Bai Y, Wang X, Shen L, Jiang K, Ding X, Cappetta D, Zhou J, Ge J, Zou Y., 2016
Exosomes derived from dendritic cells improve cardiac function via activation of CD4(+) T lymphocytes after myocardial infarction., Liu H, Gao W, Yuan J, Wu C, Yao K, Zhang L, Ma L, Zhu J, Zou Y, Ge J., 2016
Mas receptor mediates cardioprotection of angiotensin-(1-7) against Angiotensin II-induced cardiomyocyte autophagy and cardiac remodelling through inhibition of oxidative stress., Lin L, Liu X, Xu J, Weng L, Ren J, Ge J, Zou Y., 2016
High-density lipoprotein inhibits mechanical stress-induced cardiomyocyte autophagy and cardiac hypertrophy through angiotensin II type 1 receptor-mediated PI3K/Akt pathway., Lin L, Liu X, Xu J, Weng L, Ren J, Ge J, Zou Y., 2015
Timing for intracoronary administration of bone marrow mononuclear cells after acute ST-elevation myocardial infarction: a pilot study., Huang R, Yao K, Sun A, Qian J, Ge L, Zhang Y, Niu Y, Wang K, Zou Y, Ge J., 2015
Knockdown of nucleosome assembly protein 1-like 1 induces mesoderm formation and cardiomyogenesis via notch signaling in murine-induced pluripotent stem cells., Gong H, Yan Y, Fang B, Xue Y, Yin P, Li L, Zhang G, Sun X, Chen Z, Ma H, Yang C, Ding Y, Yong Y, Zhu Y, Yang H, Komuro I, Ge J, Zou Y., 2014
Olmesartan attenuates cardiac remodeling through DLL4tch1 pathway activation in pressure overload mice., You J, Wu J, Jiang G, Guo J, Wang S, Li L, Ge J, Zou Y., 2013
Comparison between adenosine and isoflurane for assessing the coronary flow reserve in mouse models of left ventricular pressure and volume overload., You J, Wu J, Ge J, Zou Y., 2012
