Professor | Chengsheng Wu | Asia Growth Partners

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Name	Chengsheng Wu
Contact Information	wuchengsheng@ucas.ac.cn
Language ability	English, Chinese
Province	Beijing

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Name

Chengsheng Wu

Contact Information

wuchengsheng@ucas.ac.cn

Language ability

English, Chinese

Province

Beijing

University/Lab Affiliation and Title	University of Chinese Academy of Sciences
Faculty	Life Sciences
Academic Background & Achievements	2018-02 to 2023-02 Postdoctoral: University of California, San Diego 2012-09 to 2017-06 PhD: University of Alberta 2009-09 to 2012-06 Master's: Fudan University 2005-09 to 2009-06 Bachelor's: Hefei University of Technology
Work Experience	2024-04 to Present - University of Chinese Academy of Sciences - Tenure-Track Assistant Professor 2023-02 to 2024-04 - University of California, San Diego - Assistant Project Scientist

University/Lab Affiliation and Title

University of Chinese Academy of Sciences

Faculty

Life Sciences

Academic Background & Achievements

2018-02 to 2023-02 Postdoctoral: University of California, San Diego
2012-09 to 2017-06 PhD: University of Alberta
2009-09 to 2012-06 Master's: Fudan University
2005-09 to 2009-06 Bachelor's: Hefei University of Technology

Work Experience

2024-04 to Present - University of Chinese Academy of Sciences - Tenure-Track Assistant Professor
2023-02 to 2024-04 - University of California, San Diego - Assistant Project Scientist

Areas of Focus	Tumor Microenvironment Non-tumor Cell Components Tumor Initiation Tumor Progression Tumor Metastasis Molecular Targets Cancer Cell Biology Molecular Biology Biochemistry Multi-omics
Publications	Tumor-initiating cells establish a niche to overcome isolation stress., First Author, 2024 Integrin αvβ3 Upregulation in Response to Nutrient Stress Promotes Lung Cancer Cell Metabolic Plasticity., Second Author, 2024 Upregulation of fibronectin and its integrin receptors - an adaptation to isolation stress that facilitates tumor initiation., First Author, 2023 Pancreatic cancer cells upregulate LPAR4 in response to isolation stress to promote an ECM-enriched niche and support tumour initiation., First Author, 2023 Metabolic Reprograming via Deletion of CISH in Human iPSC-Derived NK Cells Promotes In Vivo Persistence and Enhances Anti-tumor Activity., Eighth Author, 2020 Oxidative stress enhances tumorigenicity and stem-like features via the activation of the Wnt/β-catenin/MYC/Sox2 axis in ALK-positive anaplastic large-cell lymphoma., First Author, 2018 A positive feedback loop involving the Wnt/β-catenin/MYC/Sox2 axis defines a highly tumorigenic cell subpopulation in ALK-positive anaplastic large cell lymphoma., First Author, 2016 The PI3K/AKT/c-MYC axis promotes the acquisition of cancer stem-like features in esophageal squamous cell carcinoma., First Author, 2016 STAT1 is phosphorylated and downregulated by the oncogenic tyrosine kinase NPM-ALK in ALK-positive anaplastic large-cell lymphoma., First Author, Cover Article, 2015

Areas of Focus

Tumor Microenvironment
Non-tumor Cell Components
Tumor Initiation
Tumor Progression
Tumor Metastasis
Molecular Targets
Cancer Cell Biology
Molecular Biology
Biochemistry
Multi-omics

Publications

Tumor-initiating cells establish a niche to overcome isolation stress., First Author, 2024
Integrin αvβ3 Upregulation in Response to Nutrient Stress Promotes Lung Cancer Cell Metabolic Plasticity., Second Author, 2024
Upregulation of fibronectin and its integrin receptors - an adaptation to isolation stress that facilitates tumor initiation., First Author, 2023
Pancreatic cancer cells upregulate LPAR4 in response to isolation stress to promote an ECM-enriched niche and support tumour initiation., First Author, 2023
Metabolic Reprograming via Deletion of CISH in Human iPSC-Derived NK Cells Promotes In Vivo Persistence and Enhances Anti-tumor Activity., Eighth Author, 2020
Oxidative stress enhances tumorigenicity and stem-like features via the activation of the Wnt/β-catenin/MYC/Sox2 axis in ALK-positive anaplastic large-cell lymphoma., First Author, 2018
A positive feedback loop involving the Wnt/β-catenin/MYC/Sox2 axis defines a highly tumorigenic cell subpopulation in ALK-positive anaplastic large cell lymphoma., First Author, 2016
The PI3K/AKT/c-MYC axis promotes the acquisition of cancer stem-like features in esophageal squamous cell carcinoma., First Author, 2016
STAT1 is phosphorylated and downregulated by the oncogenic tyrosine kinase NPM-ALK in ALK-positive anaplastic large-cell lymphoma., First Author, Cover Article, 2015

Tumor Microenvironment Non-Tumor Cells Fibroblasts Immune Cells Ecm Stress Initiation Progression Metastasis

Chenxi Tian - University of Chinese Academy of Sciences

Zilong Wen - Southern University of Science and Technology

Hanjie Li - University of Chinese Academy of Sciences

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