Professor | Shuwen Zhang | Asia Growth Partners

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Name	Shuwen Zhang
Contact Information	sw.chang@siat.ac.cn
Language ability	Chinese, English, German
Province	Guangdong

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Name

Shuwen Zhang

Contact Information

sw.chang@siat.ac.cn

Language ability

Chinese, English, German

Province

Guangdong

University/Lab Affiliation and Title	University of Chinese Academy of Sciences
Faculty	Life Sciences
Work Experience	2014.1-2017.3 - Charité Hospital, Berlin, Germany - Postdoctoral Researcher 2017.7-2017.12 - Max Planck Institute, Göttingen, Germany - Postdoctoral Researcher 2019.1-2020.10 - Hospital for Sick Children, Toronto, Canada - Postdoctoral Researcher

University/Lab Affiliation and Title

University of Chinese Academy of Sciences

Faculty

Life Sciences

Work Experience

2014.1-2017.3 - Charité Hospital, Berlin, Germany - Postdoctoral Researcher
2017.7-2017.12 - Max Planck Institute, Göttingen, Germany - Postdoctoral Researcher
2019.1-2020.10 - Hospital for Sick Children, Toronto, Canada - Postdoctoral Researcher

Areas of Focus	Disordered Protein Phase Separation and Synaptic Plasticity Molecular Mechanisms of Autism Neurodevelopmental Disorders
Publications	The gut metabolite indole-3-propionic acid activates ERK1 to restore social function and hippocampal inhibitory synaptic transmission in a 16p11.2 microdeletion mouse model, Shuwen Zhang, 2024 Munc13-1 is a calcium phospholipid-dependent vesicle priming hub that shapes synaptic short-term plasticity and enables sustained neurotransmission, Shuwen Zhang, 2021 Synaptotagmin-1 drives synchronous Ca2+-triggered fusion by C2B-domain-mediated synaptic-vesicle-membrane attachment, Shuwen Zhang, 2018 Heterodimerization of Munc13 C 2 A domain with RIM regulates synaptic vesicle docking and priming, Shuwen Zhang, 2017 Distinct Functions of Syntaxin-1 in Neuronal Maintenance, Synaptic Vesicle Docking, and Fusion in Mouse Neurons, Shuwen Zhang, 2016 Complexin Stabilizes Newly Primed Synaptic Vesicles and Prevents Their Premature Fusion at the Mouse Calyx of Held Synapse, Shuwen Zhang, 2015

Areas of Focus

Disordered Protein Phase Separation and Synaptic Plasticity
Molecular Mechanisms of Autism
Neurodevelopmental Disorders

Publications

The gut metabolite indole-3-propionic acid activates ERK1 to restore social function and hippocampal inhibitory synaptic transmission in a 16p11.2 microdeletion mouse model, Shuwen Zhang, 2024
Munc13-1 is a calcium phospholipid-dependent vesicle priming hub that shapes synaptic short-term plasticity and enables sustained neurotransmission, Shuwen Zhang, 2021
Synaptotagmin-1 drives synchronous Ca2+-triggered fusion by C2B-domain-mediated synaptic-vesicle-membrane attachment, Shuwen Zhang, 2018
Heterodimerization of Munc13 C 2 A domain with RIM regulates synaptic vesicle docking and priming, Shuwen Zhang, 2017
Distinct Functions of Syntaxin-1 in Neuronal Maintenance, Synaptic Vesicle Docking, and Fusion in Mouse Neurons, Shuwen Zhang, 2016
Complexin Stabilizes Newly Primed Synaptic Vesicles and Prevents Their Premature Fusion at the Mouse Calyx of Held Synapse, Shuwen Zhang, 2015

Disordered Proteins Phase Separation Synaptic Plasticity Autism Molecular Mechanisms Neurodevelopment Synaptic Transmission Transgenic Mice Two-Photon Imaging Electrophysiology

Yuan Xiaobing - East China Normal University

Yi-Hsuan Pan - East China Normal University

Jiada Li - Central South University

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