Professor | Lei Xue | Asia Growth Partners

Name	Lei Xue
Contact Information	lxue@fudan.edu.cn
Language ability	Chinese, English
Province	Shanghai

Name

Lei Xue

Contact Information

lxue@fudan.edu.cn

Language ability

Chinese, English

Province

Shanghai

University/Lab Affiliation and Title	Fudan University
Faculty	Life Sciences
Academic Background & Achievements	2000-2005 PhD in Neurobiology: Fudan University 1996-2000 Bachelor's in Genetics: Fudan University
Work Experience	2012-present - Fudan University - Researcher, Doctoral Supervisor 2010-2012 - National Institutes of Health (NIH) - Research Scientist 2005-2010 - National Institutes of Health (NIH) - Postdoctoral Fellow
Awards	Pujiang Talent Program Shanghai Biophysical Society Young Scientific Talent

University/Lab Affiliation and Title

Fudan University

Faculty

Life Sciences

Academic Background & Achievements

2000-2005 PhD in Neurobiology: Fudan University
1996-2000 Bachelor's in Genetics: Fudan University

Work Experience

2012-present - Fudan University - Researcher, Doctoral Supervisor
2010-2012 - National Institutes of Health (NIH) - Research Scientist
2005-2010 - National Institutes of Health (NIH) - Postdoctoral Fellow

Awards

Pujiang Talent Program
Shanghai Biophysical Society Young Scientific Talent

Areas of Focus	Research on synaptic transmission function and molecular cellular mechanisms in the central nervous system, focusing on the dynamic regulation of synaptic vesicle exocytosis/endocytosis cycle, neural plasticity, and related computer modeling.
Publications	BDNF activates postsynaptic TrkB receptors to induce endocannabinoid release and inhibit presynaptic calcium influx at a calyx-type synapse., Wu YC, Liu QZ, Guo B, Ye FF, Ge JL, and Xue L., 2020 Propofol reduces synaptic strength by inhibiting sodium and calcium channels at nerve terminals., Liu QZ, Hao M, Zhou ZY, Ge JL, Wu YC, Zhao LL, Wu X, Feng Y, Gao H, Li S, and Xue L., 2019 Dynamics of transitions from anti-phase to multiple in-phase synchronizations in inhibitory coupled bursting neurons., Jia B, Wu YC, He D, Guo BH, and Xue L., 2018 Extremely Low Frequency Electromagnetic Fields Facilitate Vesicle Endocytosis by Increasing Presynaptic Calcium Channel Expression at a Central Synapse., Sun ZC, Ge JL, Guo B, Guo J, Hao M, Wu YC, Lin YA, La T, Yao PT, Mei YA, Feng Y, and Xue L., 2016 A Three-Pool Model Dissecting Readily Releasable Pool Replenishment at the Calyx of Held., Guo J, Ge JL, Hao M, Sun ZC, Wu XS, Zhu JB, Wang W, Yao PT, Lin W, and Xue L., 2015 A Monte Carlo simulation dissecting quantal release at the calyx of Held., Guo J, Sun ZC, Yao PT, Wang HL, and Xue L., 2015 Most vesicles in a central nerve terminal participate in recycling., Xue L, Sheng J, Wu XS, Wu W, Luo FJ, Shin Wonchul, Chiang HC, and Wu LG., 2013 Voltage-dependent calcium channels at the plasma membrane, but not vesicular channels, couple exocytosis to endocytosis., Xue L, Zhang Z, McNeil B, Luo FJ, Wu XS, Sheng JS, and Wu LG., 2012 A Membrane Pool Retrieved via Endocytosis Overshoot at Nerve Terminals: A Study of Its Retrieval Mechanism and Role., Xue L, McNeil BD, Wu XS, Luo FJ, He L, and Wu LG., 2012 Post-tetanic potentiation is caused by two signaling mechanisms affecting quantal size and quantal content., Xue L and Wu LG., 2010 Compound vesicle fusion increases quantal size and potentiates synaptic transmission., He L, Xue L, Xu J, McNeil B, Bai L, Melicoff E, Adachi R, and Wu LG., 2009

Areas of Focus

Research on synaptic transmission function and molecular cellular mechanisms in the central nervous system, focusing on the dynamic regulation of synaptic vesicle exocytosis/endocytosis cycle, neural plasticity, and related computer modeling.

Publications

BDNF activates postsynaptic TrkB receptors to induce endocannabinoid release and inhibit presynaptic calcium influx at a calyx-type synapse., Wu YC, Liu QZ, Guo B, Ye FF, Ge JL, and Xue L., 2020
Propofol reduces synaptic strength by inhibiting sodium and calcium channels at nerve terminals., Liu QZ, Hao M, Zhou ZY, Ge JL, Wu YC, Zhao LL, Wu X, Feng Y, Gao H, Li S, and Xue L., 2019
Dynamics of transitions from anti-phase to multiple in-phase synchronizations in inhibitory coupled bursting neurons., Jia B, Wu YC, He D, Guo BH, and Xue L., 2018
Extremely Low Frequency Electromagnetic Fields Facilitate Vesicle Endocytosis by Increasing Presynaptic Calcium Channel Expression at a Central Synapse., Sun ZC, Ge JL, Guo B, Guo J, Hao M, Wu YC, Lin YA, La T, Yao PT, Mei YA, Feng Y, and Xue L., 2016
A Three-Pool Model Dissecting Readily Releasable Pool Replenishment at the Calyx of Held., Guo J, Ge JL, Hao M, Sun ZC, Wu XS, Zhu JB, Wang W, Yao PT, Lin W, and Xue L., 2015
A Monte Carlo simulation dissecting quantal release at the calyx of Held., Guo J, Sun ZC, Yao PT, Wang HL, and Xue L., 2015
Most vesicles in a central nerve terminal participate in recycling., Xue L, Sheng J, Wu XS, Wu W, Luo FJ, Shin Wonchul, Chiang HC, and Wu LG., 2013
Voltage-dependent calcium channels at the plasma membrane, but not vesicular channels, couple exocytosis to endocytosis., Xue L, Zhang Z, McNeil B, Luo FJ, Wu XS, Sheng JS, and Wu LG., 2012
A Membrane Pool Retrieved via Endocytosis Overshoot at Nerve Terminals: A Study of Its Retrieval Mechanism and Role., Xue L, McNeil BD, Wu XS, Luo FJ, He L, and Wu LG., 2012
Post-tetanic potentiation is caused by two signaling mechanisms affecting quantal size and quantal content., Xue L and Wu LG., 2010
Compound vesicle fusion increases quantal size and potentiates synaptic transmission., He L, Xue L, Xu J, McNeil B, Bai L, Melicoff E, Adachi R, and Wu LG., 2009

Synaptic Transmission Molecular Mechanisms Central Nervous System Synaptic Vesicles Exocytosis Endocytosis Dynamic Regulation Neural Plasticity Computer Modeling Neurobiology

An Ping - China Medical University

Teng Ma - China Medical University

Genyun Chen - University of Chinese Academy of Sciences

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