Professor | Chenfang Dong | Asia Growth Partners

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Name	Chenfang Dong
Contact Information	chenfangdong@zju.edu.cn
Language ability	Chinese, English
Province	Zhejiang

Upload Avatar (500 x 500)

Name

Chenfang Dong

Contact Information

chenfangdong@zju.edu.cn

Language ability

Chinese, English

Province

Zhejiang

University/Lab Affiliation and Title	Zhejiang University
Faculty	Medical
Awards	2018: Zhejiang University Second Affiliated Hospital Research Breakthrough Award 2013: US-China Anti-Cancer Association (USCACA-NFCR) Scholar Award 2009: Chinese Preventive Medicine Association Science and Technology Progress Award Second Prize 2008: Chinese Center for Disease Control and Prevention Fourth Academic Conference Science and Technology Paper Second Prize 2007: Chinese Center for Disease Control and Prevention National Key Laboratory of Infectious Disease Prevention and Control First Youth Academic Conference Science and Technology Paper Second Prize 2007: Chinese Center for Disease Control and Prevention Science and Technology Paper Award

University/Lab Affiliation and Title

Zhejiang University

Faculty

Medical

Awards

2018: Zhejiang University Second Affiliated Hospital Research Breakthrough Award
2013: US-China Anti-Cancer Association (USCACA-NFCR) Scholar Award
2009: Chinese Preventive Medicine Association Science and Technology Progress Award Second Prize
2008: Chinese Center for Disease Control and Prevention Fourth Academic Conference Science and Technology Paper Second Prize
2007: Chinese Center for Disease Control and Prevention National Key Laboratory of Infectious Disease Prevention and Control First Youth Academic Conference Science and Technology Paper Second Prize
2007: Chinese Center for Disease Control and Prevention Science and Technology Paper Award

Areas of Focus	Oncology Breast Cancer Tumor Metabolism Tumor EMT and Tumor Stem Cells Epigenetic Modifications, Protein Lactylation Modifications
Publications	Gluconeogenesis in Cancer: Function and Regulation of PEPCK, FBPase, and G6Pase, Wang Z and Dong C, 2019 Inhibition of UGT8 Suppresses Basal-like Breast Cancer Progression by Attenuating Sulfatide-αVβ5 Axis, Cao Q, Chen X, Wu, X, Liao R, Huang P, Tan Y, Wang L, Ren G, Huang J and Dong C, 2018 Combination of novel oncolytic herpesvirus with paclitaxel as an efficient strategy for breast cancer therapy, Deng X, Shen Y, Yi M, Zhang, C, Zhao B, Zhong G, Lou W, Xue D, Leng Q, Ding J, Zhao R, Jia W, Dong C* and Dai Z, 2023 Elevated transcription and glycosylation of B3GNT5 promotes breast cancer aggressiveness, Miao Z, Cao Q, Liao R, Chen X, Li X, Bai L, Ma C, Deng X, Dai Z, Li J* and Dong C, 2022 Nuclear translocation of PLSCR1 activates STAT1 signaling in basal-like breast cancer, Huang P, Liao R, Chen X, Wu X, Li X, Wang Y, Cao Q, Dong C, 2020 AKR1B1 promotes basal-like breast cancer progression by a positive feedback loop that activates the EMT program, Wu X, Li X, Fu Q, Cao Q, Chen X, Wang M, Yu J, Long J, Yao J, Liu H, Wang D, Liao R and Dong C, 2017 Loss of ABAT-mediated GABAergic system promotes basal-like breast cancer progression by activating Ca2+-NFAT1 axis, Chen X, Cao Q, Liao R, Wu, X, Xun S, Huang J, and Dong C, 2019 IMPA2 promotes basal-like breast cancer aggressiveness by a MYC-mediated positive feedback loop, Lei X, Liao R, Chen X, Wang Z, Cao Q, Bai L, Ma C, Deng X, Ma Y, Wu X, Li J, Dai Z and Dong C, 2024 AMD1 promotes breast cancer aggressiveness via a spermidine-eIF5A hypusination-TCF4 axis, Liao R, Chen X, Cao Q, Bai L, Ma C, Dai Z and Dong C, 2024 Twist-mediated PAR1 induction is required for breast cancer progression and metastasis by inhibiting Hippo pathway, Wang Y, Liao R, Chen X, Chen G, Ying X, Chen G, Li M and Dong C, 2020 Loss of FBP1 by Snail-mediated Repression Provides Metabolic Growth Advantages in Basal-like Breast Cancer, Dong C, Yuan T, Wu Y, Wang Y, Fan T, Miriyala S, Lin Y, Yao J, Shi J, Kang T, Lorkiewicz P, Clair DS, Hung MC, Evers BM, and Zhou BP, 2013 G9a interacts with Snail and is critical for Snail‐mediated E‐cadherin repression in human breast cancer, Dong C, Wu Y, Yao J, Wang Y, Yu Y, Rychahou PG, Evers BM, and Zhou BP, 2012 Interaction with Suv39H1 is Critical for Snail‐mediated E‐cadherin Repression in Breast Cancer, Dong C, Wu Y, Wang Y, Wang C, Kang TB, Rychahou PG, Chi Y‐I, Evers BM, and Zhou BP, 2013

Areas of Focus

Oncology
Breast Cancer
Tumor Metabolism
Tumor EMT and Tumor Stem Cells
Epigenetic Modifications, Protein Lactylation Modifications

Publications

Gluconeogenesis in Cancer: Function and Regulation of PEPCK, FBPase, and G6Pase, Wang Z and Dong C*, 2019
Inhibition of UGT8 Suppresses Basal-like Breast Cancer Progression by Attenuating Sulfatide-αVβ5 Axis, Cao Q, Chen X, Wu, X, Liao R, Huang P, Tan Y, Wang L, Ren G, Huang J and Dong C*, 2018
Combination of novel oncolytic herpesvirus with paclitaxel as an efficient strategy for breast cancer therapy, Deng X, Shen Y, Yi M, Zhang, C, Zhao B, Zhong G, Lou W, Xue D, Leng Q, Ding J, Zhao R, Jia W, Dong C* and Dai Z*, 2023
Elevated transcription and glycosylation of B3GNT5 promotes breast cancer aggressiveness, Miao Z, Cao Q, Liao R, Chen X, Li X, Bai L, Ma C, Deng X, Dai Z*, Li J* and Dong C*, 2022
Nuclear translocation of PLSCR1 activates STAT1 signaling in basal-like breast cancer, Huang P, Liao R, Chen X, Wu X, Li X, Wang Y, Cao Q, Dong C*, 2020
AKR1B1 promotes basal-like breast cancer progression by a positive feedback loop that activates the EMT program, Wu X, Li X, Fu Q, Cao Q, Chen X, Wang M, Yu J, Long J, Yao J, Liu H, Wang D, Liao R and Dong C*, 2017
Loss of ABAT-mediated GABAergic system promotes basal-like breast cancer progression by activating Ca2+-NFAT1 axis, Chen X, Cao Q, Liao R, Wu, X, Xun S, Huang J, and Dong C*, 2019
IMPA2 promotes basal-like breast cancer aggressiveness by a MYC-mediated positive feedback loop, Lei X, Liao R, Chen X, Wang Z, Cao Q, Bai L, Ma C, Deng X, Ma Y, Wu X, Li J*, Dai Z* and Dong C*, 2024
AMD1 promotes breast cancer aggressiveness via a spermidine-eIF5A hypusination-TCF4 axis, Liao R, Chen X, Cao Q, Bai L, Ma C, Dai Z* and Dong C*, 2024
Twist-mediated PAR1 induction is required for breast cancer progression and metastasis by inhibiting Hippo pathway, Wang Y, Liao R, Chen X, Chen G, Ying X, Chen G, Li M and Dong C*, 2020
Loss of FBP1 by Snail-mediated Repression Provides Metabolic Growth Advantages in Basal-like Breast Cancer, Dong C, Yuan T, Wu Y, Wang Y, Fan T, Miriyala S, Lin Y, Yao J, Shi J, Kang T, Lorkiewicz P, Clair DS, Hung MC, Evers BM, and Zhou BP, 2013
G9a interacts with Snail and is critical for Snail‐mediated E‐cadherin repression in human breast cancer, Dong C, Wu Y, Yao J, Wang Y, Yu Y, Rychahou PG, Evers BM, and Zhou BP, 2012
Interaction with Suv39H1 is Critical for Snail‐mediated E‐cadherin Repression in Breast Cancer, Dong C, Wu Y, Wang Y, Wang C, Kang TB, Rychahou PG, Chi Y‐I, Evers BM, and Zhou BP, 2013

Oncology Breast Cancer Tumor Metabolism Tumor Emt Tumor Stem Cells Epigenetic Modifications Protein Lactylation Cancer Research Metabolic Reprogramming Emt Induction

Yiding Chen - Zhejiang University

Chao Ni - Zhejiang University

Fuming Qiu - Zhejiang University

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