1988-09 to 1993-06: Bachelor's degree in Preventive Medicine from Shenyang Medical College
1993-09 to 1996-06: Master's degree in Epidemiology and Statistics from Harbin Medical University
1999-07 to 2002-06: Doctorate in Pathology and Pathophysiology from First Military Medical University
1996-08 to 1999-09: Assistant and Lecturer at the Department of Epidemiology, First Military Medical University
2002-07 to 2004-08: Lecturer at the Department of Pathophysiology, First Military Medical University
2004-09 to 2006-03: Lecturer and Associate Professor at the Department of Pathophysiology, Southern Medical University
2006-03 to 2006-05: Associate Professor at the Department of Forensic Medicine, Southern Medical University
2006-05 to 2008-01: Visiting Scholar at the Department of Surgery, University of Alabama at Birmingham
2008-01 to 2016-07: Professor and Deputy Chief Forensic Physician at the Department of Forensic Medicine, Southern Medical University
2016-07 to 2021-07: Professor and Chief Forensic Physician at the School of Forensic Medicine, Southern Medical University
2021-07 to present: Professor and Chief Forensic Physician at the Experimental Management Center, Southern Medical University
2011: First Prize in Guangdong Science and Technology Award for the project 'Pathogenesis of Severe Shock'
1999: Teaching Excellence Award from First Military Medical University
2011: First Prize in Guangdong Science and Technology Award for 'Pathogenesis of Severe Shock' (7/9)
2005: Second Prize in Military Science and Technology Progress Award for 'Vascular Smooth Muscle Potassium Channel Activation and Cell Membrane Hyperpolarization in Shock' (6/9)
Research
Research on the mechanisms of trauma, burns, and hemorrhagic shock and early organ function maintenance Forensic medicine and forensic clinical identification Laboratory management research (CNAS, CMA, and scientific research laboratory accreditation)
SELECTIVE INHIBITION OF iNOS ATTENUATES TRAUMA-HEMORRHAGE/RESUSCITATION-INDUCED HEPATIC INJURY, Kan WH, Hsu JT, Schwacha MG, Choudhry MA, Raju R, Bland KI, Chaudry IH, 2008
Estrogen ameliorates trauma-hemorrhage-induced lung injury via endothelial nitric oxide synthase-dependent activation of protein kinase G, Kan WH, Hsu JT, Schwacha MG, Choudhry MA, Bland KI, Chaudry IH, 2008
The role of MIP-1alpha in the development of systemic inflammatory response and organ injury following trauma hemorrhage, Hsieh CH, Frink M, Hsieh YC, Kan WH, Hsu JT, Schwacha MG, Choudhry MA, Chaudry IH, 2008
SYSTEMATIC ANALYSIS OF THE SALUTARY EFFECT OF ESTROGEN ON CARDIAC PERFORMANCE AFTER TRAUMA-HEMORRHAGE, Ba ZF, Hsu JT, Chen J, Kan WH, Schwacha MG, Chaudry IH, 2008
Mechanism of Estrogen-Mediated Intestinal Protection following Trauma-Hemorrhage: p38 MAPK-Dependent Upregulation of HO-1, Hsu JT, Kan WH, Hsieh CH, Choudhry MA, Schwacha MG, Bland KI, Chaudry IH, 2008
MECHANISM OF ESTROGEN-MEDIATED IMPROVEMENT IN CARDIAC FUNCTION FOLLOWING TRAUMA-HEMORRHAGE: p38-DEPENDENT NORMALIZATION OF CARDIAC Akt PHOSPHORYLATION AND GLYCOGEN LEVELS, Hsu JT, Kan WH, Hsieh YC, Choudhry MA, Schwacha MG, Bland KI, Chaudry IH, 2008
p38 MAPK-Dependent eNOS Upregulation is Critical for 17 beta-estradiol-mediated Cardioprotection following trauma-hemorrhage, Kan WH, Hsu JT, Ba ZF, Schwacha MG, Chen JG, Choudhry MA, Bland KI, Chaudry IH, 2008
Flutamide protects against trauma-hemorrhage-induced liver injury via attenuation of the inflammatory response, oxidative stress and apopotosis, Kan WH, Hsieh CH, Schwacha MG, Choudhry MA, Raju R, Bland KI, Chaudry IH, 2008
Mechanism of estrogen-mediated attenuation of hepatic injury following trauma-hemorrhage: Akt-dependent HO-1 up-regulation, Hsu, J.T.; Kan, W.H.; Hsieh, C.H.; Choudhry, M.A.; Schwacha, M.G.; Bland, K.I.; Chaudry, I.H., 2007
Role of p38 mitogen-activated protein kinase pathway in estrogen-mediated cardioprotection following trauma-hemorrhage, Hsu JT, Hsieh YC, Kan WH, Chen JG, Choudhry MA, Schwacha MG, Bland KI, Chaudry IH, 2007
17b-estradiol-mediated cardioprotection following trauma-hemorrhage involves activation of p38 MAP kinase and eNOS, W.H. Kan, J.T. Hsu, Z.F.Ba, M.G. Schwacha, M.A. Choudhry, K.I. Bland, and I. H. Chaudry, 2007
Mechanism of estrogen-mediated cardioprotection following trauma-hemorrhage: p38/Akt-dependent increase in cardiac glucose levels, J.T. Hsu, W.H. Kan, Y.C. Hsieh, M.A. Choudhry, M.G. Schwacha, K.I. Bland, and I. H. Chaudry, 2007
The role of MIP-1a in the systemic inflammatory response and lung injury following trauma-hemorrhage, C.H. Hsieh, M. Frink, Y. C. Hsieh, W.H. Kan, M.G. Schwacha and I. H. Chaudry, 2007
The development of mitochondrial nitric oxide synthase study, Kan Wenhong, Zhao Keseng, 2006
Mitochondrial nitric oxide synthase in the inflammation and septic shock, Kan Wenhong, 2006
The effect of mitochondrial nitric oxide synthase on inflammation and septic shock, Kan wenhong, 2005
Lung, spleen, and kidney are the major places for inducible nitric oxide synthase expression in endotoxic shock: role of p38 mitogen-activated protein kinase in signal transduction of inducible nitric oxide synthase expression, Kan Wenhong, Zhao KS, Jiang Y, Yan W, Huang Q, Wang J, Qin Q, Huang X, Wang S, 2004
Role of p38 MAPK in ICAM-1 expression of vascular endothelial cells induced by lipopolysaccharide, WS Yan, K Zhao, Y Jiang, Q Huang, J Wang, W Kan, and S Wang, 2002
Expression of intercellular adhesion molecule-1 in different organs of the mice with endotoxic shock induced by lipopolysaccharide, YAN WenSheng , KAN Wen Hong , HUANG Qiao Bing , JIANG Yong , WANG Shi Wen , ZHAO Ke Seng, 2002
Role of p38 MAPK in ICAM-1 expression of vascular endothelial cells induced by lipopolysaccharide, WS Yan, K Zhao, Y Jiang, Q Huang, J Wang, W Kan, and S Wang, 2002
Role of p38 MAPK in lipopolysaccharide-induced iNOS expression by endothelial cells, KAN Wen-hong, YAN Wen-sheng, JIANG Yong, WANG Jing-zhen, QIN Qing-he, ZHAO Ke-seng, 2002
Area of Focus
Immune Regulation | Molecular Mechanisms | T Cell Development | Signal Transduction | Toll-Like Receptors | Cd8 Regulatory T Cells | Autoimmune Diseases | Pathogenesis | Treatment | Research
