Professor | Junshuang Jia | Asia Growth Partners

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Name	Junshuang Jia
Contact Information	jiajsh4@smu.edu.cn
Language ability	Chinese, English
Province	Guangdong

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Name

Junshuang Jia

Contact Information

jiajsh4@smu.edu.cn

Language ability

Chinese, English

Province

Guangdong

University/Lab Affiliation and Title	Southern Medical University
Faculty	Basic Medical Sciences
Academic Background & Achievements	2005.9–2008.7 Master's Degree in Pathology and Pathophysiology: Southern Medical University
Work Experience	2008.7-2012.12 - Southern Medical University, Basic Medical College, Cancer Research Institute - Assistant Experimentalist 2013.1-2018.11 - Southern Medical University, Basic Medical College, Cancer Research Institute - Experimentalist 2018.12-present - Southern Medical University, Basic Medical College, Cancer Research Institute - Senior Experimentalist

University/Lab Affiliation and Title

Southern Medical University

Faculty

Basic Medical Sciences

Academic Background & Achievements

2005.9–2008.7 Master's Degree in Pathology and Pathophysiology: Southern Medical University

Work Experience

2008.7-2012.12 - Southern Medical University, Basic Medical College, Cancer Research Institute - Assistant Experimentalist
2013.1-2018.11 - Southern Medical University, Basic Medical College, Cancer Research Institute - Experimentalist
2018.12-present - Southern Medical University, Basic Medical College, Cancer Research Institute - Senior Experimentalist

Areas of Focus	Tumor Research Preparation of Genetically Engineered Animals
Publications	miR-155 enhances insulin sensitivity by coordinated regulation of multiple genes in mice., Lin XL, Qin YJ, Jia JS, et al., 2016 Ectopic expression of Cripto-1 in transgenic mouse embryos causes hemorrhages, fatal cardiac defects and embryonic lethality., Lin XL, Zhao WT, Jia JS, et al., 2016 R/L, a double reporter mouse line that expresses luciferase gene upon Cre-mediated excision, followed by inactivation of mRFP expression., Jia JS, Lin XL, et al., 2016 Targeted disruption of miR-17-92 impairs mouse spermatogenesis by activating mTOR signaling pathway., Xie RY, Lin XL, Du T, et al., 2016 Simple and rapid determination of homozygous transgenic mice via in vivo fluorescence imaging., Lin XL, Jia JS, Qin YJ, et al., 2015 Overexpression of miR-155 in the liver of transgenic mice alters the expression profiling of hepatic genes associated with lipid metabolism., Lin XL, Jia JS, Du T, et al., 2015 Generation of Rm21LG transgenic mice: a powerful tool to generate conditional overexpression of miR-21 that is involved in oncogenesis., Du T, Jia JS, Lin XL, et al., 2014 Klf4 reduces stemness phenotype, triggers mesenchymal-epithelial transition (MET)-like molecular changes, and prevents tumor progression in nasopharygeal carcinoma., LiXQ, Zhao ZL, Zhang XL, et al., 2017

Areas of Focus

Tumor Research
Preparation of Genetically Engineered Animals

Publications

miR-155 enhances insulin sensitivity by coordinated regulation of multiple genes in mice., Lin XL, Qin YJ, Jia JS, et al., 2016
Ectopic expression of Cripto-1 in transgenic mouse embryos causes hemorrhages, fatal cardiac defects and embryonic lethality., Lin XL, Zhao WT, Jia JS, et al., 2016
R/L, a double reporter mouse line that expresses luciferase gene upon Cre-mediated excision, followed by inactivation of mRFP expression., Jia JS, Lin XL, et al., 2016
Targeted disruption of miR-17-92 impairs mouse spermatogenesis by activating mTOR signaling pathway., Xie RY, Lin XL, Du T, et al., 2016
Simple and rapid determination of homozygous transgenic mice via in vivo fluorescence imaging., Lin XL, Jia JS, Qin YJ, et al., 2015
Overexpression of miR-155 in the liver of transgenic mice alters the expression profiling of hepatic genes associated with lipid metabolism., Lin XL, Jia JS, Du T, et al., 2015
Generation of Rm21LG transgenic mice: a powerful tool to generate conditional overexpression of miR-21 that is involved in oncogenesis., Du T, Jia JS, Lin XL, et al., 2014
Klf4 reduces stemness phenotype, triggers mesenchymal-epithelial transition (MET)-like molecular changes, and prevents tumor progression in nasopharygeal carcinoma., LiXQ, Zhao ZL, Zhang XL, et al., 2017

Transgenosis Gene Knockout Oncogenesis Tumor Mechanism Immunotherapy Genetic Engineering Animal Models Cancer Research Biomedical Research Experimental Techniques

Huanran Tan - Peking University

Lu Jing - Capital Medical University

Mingxi Liu - Nanjing Medical University

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