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Cong Xin
congxin@bjmu.edu.cn
Chinese, English
Beijing
Peking University
Basic Medical Sciences
  • 2007 - Bachelor of Medicine: Peking University
  • 2012 - PhD in Pathophysiology: Peking University
  • Postdoctoral work at Peking University Medical Department from 2012 to 2014
  • 2012 - 2014 - Peking University Medical Department, Postdoctoral Researcher
  • Current - Peking University, Associate Professor
  • 2015 - Second Prize of Science and Technology Progress by Ministry of Education
  • 2016 - First Prize of Huaxia Medical Award
  • 2016 - First Prize at the International Physiology Conference Young Scientist Session
  • 2015 - First Prize at the National Salivary Gland Disease Academic Seminar
  • 2015 - Second Prize at the 10th National Congress of Pathophysiology in China
Regulation mechanisms of intercellular junctions
  • Interleukin-13 promotes cellular senescence through inducing mitochondrial dysfunction in IgG4-related sialadenitis, Zhu M, Min S, Mao X, Zhou Y, Zhang Y, Li W, Li L, Wu L, Cong X, Yu G
  • Matricellular proteins: potential biomarkers and mechanistic factors in aortic aneurysms, Li Z, Cong X, Kong W, 2022
  • In vivo vascular permeability assessment in mouse submandibular gland, Mao X, Min S, He Q, Cong X
  • Contribution of interleukin-4-induced epithelial cell senescence to glandular fibrosis in IgG4-related sialadenitis, Min SN, Zhu MQ, Mao XD, Li W, Wei T, Mei M, Zhang Y, Wu LL, Yu GY, Cong X, 2022
  • Disruption of tight junction structure contributes to secretory dysfunction in IgG4-related sialadenitis, Min SN, Wu LL, Zhang YY, Zhu WX, Cong X, Yu GY, 2020
  • Muscarinic acetylcholine receptor-mediated tight junction opening is involved in epiphora in late phase of submandibular gland transplantation, Yang NY, Ding C, Li J, Zhang Y, Xiang RL, Wu LL, Yu GY, Cong X, 2017
  • Endothelial tight junctions and their regulatory signaling pathways in vascular homeostasis and disease, Cong X, Kong W, 2020
  • Tricellulin modulates transport of macromolecules in the salivary gland, Min SN, Cong X, Zhang Y, Xiang RL, Zhou Y, Yu GY, Wu LL, 2020
  • Disruption of endothelial barrier function is linked with hyposecretion and lymphocytic infiltration in salivary glands of Sjögren's syndrome, Cong X, Zhang XM, Zhang Y, Wei T, He QH, Zhang LW, Hua H, Lee SW, Park K, Yu GY, Wu LL, 2018
  • Decreased interaction between ZO-1 and occludin is involved in alteration of tight junction in transplanted epiphora submandibular glands, Ding C, Cong X, Zhang XM, Li SL, Wu LL, Yu GY, 2017
  • Parasympathectomy increases resting salivary secretion in normal and irradiated submandibular glands of rats, Qi W, Cong X, Zhang XM, Wang ZY, Yang NY, Ding C, Shan XF, Wu LL, Yu GY, 2017
  • Endothelial tight junctions are opened in cholinergic-evoked salivation in vivo, Cong X, Zhang Y, He QH, Wei T, Zhang XM, Zhang JZ, Xiang RL, Yu GY, Wu LL, 2017
  • Interleukin-17 impairs salivary tight junction integrity in Sjögren's syndrome, Zhang LW, Cong X, Zhang Y, Wei T, Su YC, Serrão AC, Brito AR Jr, Yu GY, Hua H, Wu LL, 2016
  • Claudin-4 is required for modulation of paracellular permeability by muscarinic acetylcholine receptor in epithelial cells, Cong X, Zhang Y, Li J, Mei M, Ding C, Xiang RL, Zhang LW, Wang Y, Wu LL, Yu GY, 2015
  • Occludin is required for TRPV1-modulated paracellular permeability in the submandibular gland, Cong X, Zhang Y, Yang NY, Li J, Ding C, Ding QW, Su YC, Mei M, Guo XH, Wu LL, Yu GY, 2013
  • Activation of transient receptor potential vanilloid subtype 1 increases expression and permeability of tight junction in normal and hyposecretory submandibular gland, Cong X, Zhang Y, Shi L, Yang NY, Ding C, Li J, Ding QW, Su YC, Xiang RL, Wu LL, Yu GY, 2012
  • Activation of transient receptor potential vanilloid subtype 1 increase secretion of the hypofunctional, transplanted submandibular gland, Zhang Y, Cong X, Shi L, Xiang B, Li YM, Ding QW, Ding C, Wu LL, Yu GY, 2010
Intercellular Junctions Regulation Mechanisms Cell Biology Pathophysiology Epithelial Cells Vascular Endothelium Salivary Glands Tight Junction Proteins

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