Professor | Zhong Chao | Asia Growth Partners

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Name	Zhong Chao
Contact Information	zhongchaobjmu@hotmail.com
Language ability	Chinese, English
Province	Beijing

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Name

Zhong Chao

Contact Information

zhongchaobjmu@hotmail.com

Language ability

Chinese, English

Province

Beijing

University/Lab Affiliation and Title	Peking University
Faculty	Basic Medical Sciences
Academic Background & Achievements	2016 - PhD: Institute of Biophysics, Chinese Academy of Sciences 2002 - Bachelor: University of Science and Technology of China
Work Experience	2016 - Present: Researcher at Peking University Health Science Center 2012 - 2016: Postdoctoral Fellow at the National Institutes of Health, USA 2006 - 2012: PhD Candidate at the Institute of Biophysics, Chinese Academy of Sciences

University/Lab Affiliation and Title

Peking University

Faculty

Basic Medical Sciences

Academic Background & Achievements

2016 - PhD: Institute of Biophysics, Chinese Academy of Sciences
2002 - Bachelor: University of Science and Technology of China

Work Experience

2016 - Present: Researcher at Peking University Health Science Center
2012 - 2016: Postdoctoral Fellow at the National Institutes of Health, USA
2006 - 2012: PhD Candidate at the Institute of Biophysics, Chinese Academy of Sciences

Areas of Focus	Systems biology approaches to transcription and metabolic regulation in the immune system Key regulatory molecules in mucosal immune system homeostasis and disease New mechanisms of tumor immunity affecting cancer development and treatment
Publications	IL-21R-STAT3 signalling initiates a differentiation program in uterine tissue-resident NK cells to support pregnancy, Zhong C et al., 2023 Proline uptake promotes activation of lymphoid tissue inducer cells to maintain gut homeostasis, Zhong C et al., 2023 Genetic distinction between functional tissue-resident and conventional natural killer cells, Zhong C et al., 2023 PD-1 signaling facilitates LTi activation through restraining fatty acid oxidation, Zhong C et al., 2022 Proximal and distal regions of pathogenic Th17 related chromatin loci are sequentially accessible during pathogenicity of Th17, Zhong C et al., 2022 Deciphering cell type-specific gene expression signatures of cardiac diseases through reconstruction of bulk transcriptomes, Zhong C et al., 2022 Differential Expression of the Transcription Factor GATA3 Specifies Lineage and Functions of Innate Lymphoid Cells, Zhong C et al., 2020 Group 3 innate lymphoid cells continuously require the transcription factor GATA-3 after commitment, Zhong C et al., 2016 The transcription factor GATA3 is critical for the development of all IL-7Rα-expressing innate lymphoid cells, Zhong C et al., 2014 Granzyme K inhibits replication of influenza virus through cleaving the nuclear transport complex importin α1/β dimer of infected host cells, Zhong C et al., 2012

Areas of Focus

Systems biology approaches to transcription and metabolic regulation in the immune system
Key regulatory molecules in mucosal immune system homeostasis and disease
New mechanisms of tumor immunity affecting cancer development and treatment

Publications

IL-21R-STAT3 signalling initiates a differentiation program in uterine tissue-resident NK cells to support pregnancy, Zhong C et al., 2023
Proline uptake promotes activation of lymphoid tissue inducer cells to maintain gut homeostasis, Zhong C et al., 2023
Genetic distinction between functional tissue-resident and conventional natural killer cells, Zhong C et al., 2023
PD-1 signaling facilitates LTi activation through restraining fatty acid oxidation, Zhong C et al., 2022
Proximal and distal regions of pathogenic Th17 related chromatin loci are sequentially accessible during pathogenicity of Th17, Zhong C et al., 2022
Deciphering cell type-specific gene expression signatures of cardiac diseases through reconstruction of bulk transcriptomes, Zhong C et al., 2022
Differential Expression of the Transcription Factor GATA3 Specifies Lineage and Functions of Innate Lymphoid Cells, Zhong C et al., 2020
Group 3 innate lymphoid cells continuously require the transcription factor GATA-3 after commitment, Zhong C et al., 2016
The transcription factor GATA3 is critical for the development of all IL-7Rα-expressing innate lymphoid cells, Zhong C et al., 2014
Granzyme K inhibits replication of influenza virus through cleaving the nuclear transport complex importin α1/β dimer of infected host cells, Zhong C et al., 2012

Systems Biology Transcription Metabolic Regulation Immune System Mucosal Immunity Homeostasis Disease Process Tumor Immunity Cancer Development Cancer Treatment

Qi Sun - Peking University

Xiaojian Wang - Zhejiang University

Huang Bo - Peking Union Medical College

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