Professor | Liang Weng | Asia Growth Partners

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Name	Liang Weng
Contact Information	wengliang@bjmu.edu.cn
Language ability	English, Chinese, Japanese
Province	Beijing

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Name

Liang Weng

Contact Information

wengliang@bjmu.edu.cn

Language ability

English, Chinese, Japanese

Province

Beijing

University/Lab Affiliation and Title	Peking University
Faculty	Basic Medical Sciences
Academic Background & Achievements	2005 B.S. in Basic Medical Science: Peking University 2009 M.S. in Geriatrics: Beijing Institute of Geriatrics, National Health Commission 2013 Ph.D. in Pathology: Nagoya University
Work Experience	2013-2014 - Postdoctoral Fellow, Department of Pathology, Nagoya University, Japan 2014-2018 - Designated Assistant Professor, Department of Pathology, Nagoya University, Japan 2018-2024 - Associate Investigator, Xiangya Hospital, Central South University, China 2024-present - Assistant Professor (Tenure-Track), Associate Investigator, Department of Pathology, School of Basic Medical Science, Peking University Health Science Center, China

University/Lab Affiliation and Title

Peking University

Faculty

Basic Medical Sciences

Academic Background & Achievements

2005 B.S. in Basic Medical Science: Peking University
2009 M.S. in Geriatrics: Beijing Institute of Geriatrics, National Health Commission
2013 Ph.D. in Pathology: Nagoya University

Work Experience

2013-2014 - Postdoctoral Fellow, Department of Pathology, Nagoya University, Japan
2014-2018 - Designated Assistant Professor, Department of Pathology, Nagoya University, Japan
2018-2024 - Associate Investigator, Xiangya Hospital, Central South University, China
2024-present - Assistant Professor (Tenure-Track), Associate Investigator, Department of Pathology, School of Basic Medical Science, Peking University Health Science Center, China

Areas of Focus	Metabolic reprogramming and immune microenvironment remodeling in disease progression, especially in tumors Mechanisms of lung cancer occurrence and resistance to immunotherapy
Publications	Endothelial RIPK1 protects artery bypass graft against arteriosclerosis by regulating SMC growth, Liang Weng, 2023 E3 ligase Trim35 inhibits LSD1 demethylase activity through K63-linked ubiquitination and enhances anti-tumor immunity in NSCLC, Liang Weng, 2023 Regulation of cargo-selective endocytosis by dynamin 2 GTPase-activating protein girdin, Liang Weng, 2014 Negative regulation of amino acid signaling by MAPK-regulated 4F2hc/Girdin complex, Liang Weng, 2018 Identification of tumour-infiltrating myeloid subsets associated with overall survival in lung squamous cell carcinoma, Liang Weng, 2023 Assessment of causal effects of visceral adipose tissue on risk of cancers: a Mendelian randomization study, Liang Weng, 2022 Paradoxical effects of DNA tumor virus oncogenes on epithelium-derived tumor cell fate during tumor progression and chemotherapy response, Liang Weng, 2021 SLC3A2 Promotes Tumor Associated Macrophage Polarization via Metabolic Reprogramming in Lung Cancer, Liang Weng, 2023 Girdin Promotes Tumorigenesis and Chemoresistance in Lung Adenocarcinoma by Interacting with PKM2, Liang Weng, 2022 Dvl2 facilitates the coordination of NF-κB and Wnt signaling to promote colitis-associated colorectal progression, Liang Weng, 2022

Areas of Focus

Metabolic reprogramming and immune microenvironment remodeling in disease progression, especially in tumors
Mechanisms of lung cancer occurrence and resistance to immunotherapy

Publications

Endothelial RIPK1 protects artery bypass graft against arteriosclerosis by regulating SMC growth, Liang Weng, 2023
E3 ligase Trim35 inhibits LSD1 demethylase activity through K63-linked ubiquitination and enhances anti-tumor immunity in NSCLC, Liang Weng, 2023
Regulation of cargo-selective endocytosis by dynamin 2 GTPase-activating protein girdin, Liang Weng, 2014
Negative regulation of amino acid signaling by MAPK-regulated 4F2hc/Girdin complex, Liang Weng, 2018
Identification of tumour-infiltrating myeloid subsets associated with overall survival in lung squamous cell carcinoma, Liang Weng, 2023
Assessment of causal effects of visceral adipose tissue on risk of cancers: a Mendelian randomization study, Liang Weng, 2022
Paradoxical effects of DNA tumor virus oncogenes on epithelium-derived tumor cell fate during tumor progression and chemotherapy response, Liang Weng, 2021
SLC3A2 Promotes Tumor Associated Macrophage Polarization via Metabolic Reprogramming in Lung Cancer, Liang Weng, 2023
Girdin Promotes Tumorigenesis and Chemoresistance in Lung Adenocarcinoma by Interacting with PKM2, Liang Weng, 2022
Dvl2 facilitates the coordination of NF-κB and Wnt signaling to promote colitis-associated colorectal progression, Liang Weng, 2022

Metabolic Reprogramming Immune Microenvironment Disease Progression Tumors Lung Cancer Immunotherapy Resistance Mechanisms Post-Translational Modifications Subtype Transition Diagnostic Markers

Guizhong Cui - Guangzhou Medical University

Xiaoyan Qiu - Peking University

Ji-Min Shao - Zhejiang University

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