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Biwen Peng
pengbiwen@whu.edu.cn
English, Chinese
Hubei
Wuhan University
Medical School
  • 1992.9-1997.7 Bachelor of Clinical Medicine, Hubei Medical University
  • 1997.9-2000.7 Master of Physiology, Wuhan University Medical School
  • 2000.9-2003.7 PhD in Pathogenic Biology, Wuhan University Medical School
  • 2004.12-2007.2 Postdoctoral Fellow in Pharmacology, University of Texas Medical Center
  • 1997.7-2002.10 Assistant, Department of Physiology, Wuhan University Medical School
  • 2002.10-2004.11 Lecturer, Department of Physiology, Wuhan University Medical School
  • 2008.10-2013.10 Associate Professor, Department of Physiology, Wuhan University Medical School
  • 2013.11-present Professor, Department of Physiology, Wuhan University Medical School
  • 2007 Fujian Provincial People's Government Science and Technology Third Prize
  • 2011 11th Zhang Xijun Fund National Young Outstanding Physiology Academic Paper Exchange and Excellent Poster Award
  • 2011 Second Wuhan Natural Science Excellent Academic Paper Award Second Prize
  • 2013 Chinese Physiology Society Young Scientist Travel Award for IUPS (Birmingham, UK)
  • 2020 Hubei Colleges and Universities First-Class Undergraduate Course Award for 'The Mystery of Life - Essentials of Human Physiology'
  • 2017-2018 Wuhan University Undergraduate Excellent Teaching Performance Award, Second 'Cha Quanxing Professor 1977 Teaching Award'
  • Wuhan University 13th 'Respect Teachers and Love Learning - My Favorite Top Ten Excellent Teachers'
Neuroimmune mechanisms of paroxysmal neurological disorders
Effects of glial cells on synaptic activity and mechanisms, especially the role of microglia in synapse formation and neuronal activity
Mechanisms of active substance release by microglia, providing insights into mechanisms of neonatal epilepsy, hypoxic ischemic and thermal stress injuries
  • Complement C3 Aggravates Post-epileptic Neuronal Injury Via Activation of TRPV1, Peng BW et al., 2021
  • Silencing of circIgf1r plays a protective role in neuronal injury via regulating astrocyte polarization during epilepsy, Peng BW et al., 2021
  • NFAT5 and HIF-1α Coordinate to Regulate NKCC1 Expression in Hippocampal Neurons After Hypoxia-Ischemia, Peng BW et al., 2019
  • TRPV1 mediates astrocyte activation and interleukin-1β release induced by hypoxic ischemia (HI), Peng BW et al., 2019
  • TRPV1 translocated to astrocytic membrane to promote migration and inflammatory infiltration thus promotes epilepsy after hypoxic ischemia in immature brain, Peng BW et al., 2019
  • Activation of TRPV1 Contributes to Recurrent Febrile Seizures via Inhibiting the Microglial M2 Phenotype in the Immature Brain, Peng BW et al., 2019
  • Vitexin reduces epilepsy after hypoxic ischemia in the neonatal brain via inhibition of NKCC1, Peng BW et al., 2018
  • Modulation of neuroinflammation: Role and therapeutic potential of TRPV1 in the neuro-immune axis, Peng BW et al., 2017
Neuroimmune Paroxysmal Disorders Glial Cells Synaptic Activity Microglia Synapse Formation Neuronal Activity Active Substance Release Neonatal Epilepsy Hypoxic Ischemic

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