Professor | Hui Li | Asia Growth Partners

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Name	Hui Li
Contact Information	huili78@fudan.edu.cn
Language ability	Chinese, English
Province	Shanghai

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Name

Hui Li

Contact Information

huili78@fudan.edu.cn

Language ability

Chinese, English

Province

Shanghai

University/Lab Affiliation and Title	Fudan University
Faculty	Basic Medical Sciences
Awards	Fudan University Shanghai Medical College 'Research Scholar' Talent Program (2007) Advanced Individual in Technical Platform Service, Basic Medical College of Fudan University (2016)

University/Lab Affiliation and Title

Fudan University

Faculty

Basic Medical Sciences

Awards

Fudan University Shanghai Medical College 'Research Scholar' Talent Program (2007)
Advanced Individual in Technical Platform Service, Basic Medical College of Fudan University (2016)

Areas of Focus	Prevention and treatment mechanisms of diabetes and its complications Oxidative stress and antioxidant mechanisms Energy metabolism regulation mechanisms
Publications	HGF protected against diabetic nephropathy via autophagy-lysosome pathway in podocyte by modulating PI3K/Akt-GSK3β-TFEB axis, Hou B, Li Y, Li X, Zhang C, Zhao Z, Chen Q, Zhang N, Li H, 2020 HGF alleviates high glucose-induced injury in podocytes by GSK3β inhibition and autophagy restoration, Zhang C, Hou B, Yu S, Chen Q, Zhang N, Li H, 2016 Sulforaphane attenuation of experimental diabetic nephropathy involves GSK-3 beta/Fyn/Nrf2 signaling pathway, Shang G, Tang X, Gao P, Guo F, Liu H, Zhao Z, Chen Q, Jiang T, Zhang N, Li H, 2015 3, 5-Diiodo-L-thyronine ameliorates diabetic nephropathy in streptozotocin-induced diabetic rats, Shang G, Gao P, Zhao Z, Chen Q, Jiang T, Zhang N, Li H, 2013 HGF-mediated inhibition of oxidative stress by 8-nitro-cGMP in high glucose-treated rat mesangial cells, Guoguo S, Akaike T, Tao J, Qi C, Nong Z, Hui L, 2012 HGF Suppresses high glucose-mediated oxidative stress in mesangial cells by activation of PKG and inhibition of PKA, Li H, Yu H, Zhao JJ, Huang Y, Chen Q, Zhang N, 2010

Areas of Focus

Prevention and treatment mechanisms of diabetes and its complications
Oxidative stress and antioxidant mechanisms
Energy metabolism regulation mechanisms

Publications

HGF protected against diabetic nephropathy via autophagy-lysosome pathway in podocyte by modulating PI3K/Akt-GSK3β-TFEB axis, Hou B, Li Y, Li X, Zhang C, Zhao Z, Chen Q, Zhang N, Li H*, 2020
HGF alleviates high glucose-induced injury in podocytes by GSK3β inhibition and autophagy restoration, Zhang C, Hou B, Yu S, Chen Q, Zhang N, Li H*, 2016
Sulforaphane attenuation of experimental diabetic nephropathy involves GSK-3 beta/Fyn/Nrf2 signaling pathway, Shang G, Tang X, Gao P, Guo F, Liu H, Zhao Z, Chen Q, Jiang T, Zhang N, Li H*, 2015
3, 5-Diiodo-L-thyronine ameliorates diabetic nephropathy in streptozotocin-induced diabetic rats, Shang G, Gao P, Zhao Z, Chen Q, Jiang T, Zhang N, Li H*, 2013
HGF-mediated inhibition of oxidative stress by 8-nitro-cGMP in high glucose-treated rat mesangial cells, Guoguo S, Akaike T, Tao J, Qi C, Nong Z, Hui L*, 2012
HGF Suppresses high glucose-mediated oxidative stress in mesangial cells by activation of PKG and inhibition of PKA, Li H*, Yu H, Zhao JJ, Huang Y, Chen Q, Zhang N, 2010

Diabetes Complications Prevention Treatment Oxidative Stress Antioxidant Energy Metabolism Regulation Mechanisms Health

Youe Yan - Wuhan University

Yaxi Zhu - Central South University

Xiangjun Zeng - Capital Medical University

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