Professor | Baojie Li | Asia Growth Partners

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Name	Baojie Li
Contact Information	libj@sjtu.edu.cn
Language ability	Chinese, English
Province	Shanghai

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Name

Baojie Li

Contact Information

libj@sjtu.edu.cn

Language ability

Chinese, English

Province

Shanghai

University/Lab Affiliation and Title	Shanghai Jiao Tong University
Faculty	Bio-X Research Institute
Academic Background & Achievements	1984-1988 Bachelor: Beijing Normal University 1992-1994 Master: Albert Einstein College of Medicine 1992-1997 Doctorate: Albert Einstein College of Medicine 1997-2001 Postdoctoral: HHMI, Columbia University Changjiang Scholar Distinguished Professor Shanghai Thousand Talents Program Distinguished Professor
Work Experience	Before returning to China - Institute of Molecular and Cell Biology, Singapore - Principal Investigator (Assistant, Associate Professor) Current - Shanghai Jiao Tong University Bio-X Institute - Vice Dean and Director of Stem Cell and Developmental Research Center

University/Lab Affiliation and Title

Shanghai Jiao Tong University

Faculty

Bio-X Research Institute

Academic Background & Achievements

1984-1988 Bachelor: Beijing Normal University
1992-1994 Master: Albert Einstein College of Medicine
1992-1997 Doctorate: Albert Einstein College of Medicine
1997-2001 Postdoctoral: HHMI, Columbia University
Changjiang Scholar Distinguished Professor
Shanghai Thousand Talents Program Distinguished Professor

Work Experience

Before returning to China - Institute of Molecular and Cell Biology, Singapore - Principal Investigator (Assistant, Associate Professor)
Current - Shanghai Jiao Tong University Bio-X Institute - Vice Dean and Director of Stem Cell and Developmental Research Center

Areas of Focus	Regulation mechanisms of mesenchymal stem cell self-renewal, differentiation, and aging Mechanisms of skeletal development, bone metabolism, and osteoporosis DNA damage response, aging, and tumorigenesis
Publications	S6K1 is a multifaceted regulator of Mdm2 that connects nutrient status and DNA damage response., Keng Po Lai, Wai Fook Leong, Jenny Fung Ling Chau, Deyong Jia, Li Zeng, Huijuan Liu, Aijun Hao, Lin He, Hongbing Zhang, David Meek, Chakradhar Velagapudi, Samy L. Habib, and Baojie Li, 2010 Atypical Atm-p53 interaction in osteogenesis is mediated by Smad1 signaling., Gang Ma, Lili Li, Yuanyu Hu, Bijin Au, Lin He and Baojie Li, 2012 A crucial role for BMP-Smad1 signaling in the DNA damage response., Jenny Chau, Yuanyu Hu, Jianhe Wang, Zhi Liu, Bi Jin Au, Yuji Mishina, Ye-Guang Chen, Christine Biondi, Elizabeth Robertson, Yue-Zheng Du, Dong Xie, Xueying Wang, Qiang Yu, and Baojie Li, 2012 c-Abl promotes osteoblast expansion by differentially regulating canonical and non-canonical BMP pathways and p16(INK4a) expression., Kua HY, Liu H, Leong WF, Li L, Jia D, Ma G, Hu Y, Wang X, Chau JF, Chen YG, Mishina Y, Boast S, Yeh J, Xia L, Chen GQ, He L, Goff SP, and Baojie Li, 2012 p53 regulates neural stem cell self-renewal and differentiation via BMP-Smad1-Id1 pathway., Huijuan Liu, Deyong Jia, Jenny Chau, Lin He and Baojie Li, 2013 A critical role for p57Kip2 in the DNA damage response., Hao Jia, Jenny Fang Ling Chau, Qian Cong, and Baojie Li, 2014 Ablation of Tak1 in monocyte leads to defects in skeletal growth and bone remodeling in mice., Bing Qi, Qian Cong, Ping Li, Gang Ma, Xizhi Guo, Min Xie, Michael D. Schneider, Huijuan Liu, and Baojie Li, 2014 p53 deficiency-induced Smad1 up-regulation suppresses tumorigenesis and causes chemoresistance in colorectal cancers., Xinsen Ruan, Qiao Zuo, Jenny Chua, Hao Jia, Jinlin Lin, Xuechun Xia, Huijuan Liu, Samy L. Habib, Chuangang Fu, and Baojie Li, 2015

Areas of Focus

Regulation mechanisms of mesenchymal stem cell self-renewal, differentiation, and aging
Mechanisms of skeletal development, bone metabolism, and osteoporosis
DNA damage response, aging, and tumorigenesis

Publications

S6K1 is a multifaceted regulator of Mdm2 that connects nutrient status and DNA damage response., Keng Po Lai, Wai Fook Leong, Jenny Fung Ling Chau, Deyong Jia, Li Zeng, Huijuan Liu, Aijun Hao, Lin He, Hongbing Zhang, David Meek, Chakradhar Velagapudi, Samy L. Habib, and Baojie Li, 2010
Atypical Atm-p53 interaction in osteogenesis is mediated by Smad1 signaling., Gang Ma, Lili Li, Yuanyu Hu, Bijin Au, Lin He and Baojie Li, 2012
A crucial role for BMP-Smad1 signaling in the DNA damage response., Jenny Chau, Yuanyu Hu, Jianhe Wang, Zhi Liu, Bi Jin Au, Yuji Mishina, Ye-Guang Chen, Christine Biondi, Elizabeth Robertson, Yue-Zheng Du, Dong Xie, Xueying Wang, Qiang Yu, and Baojie Li, 2012
c-Abl promotes osteoblast expansion by differentially regulating canonical and non-canonical BMP pathways and p16(INK4a) expression., Kua HY, Liu H, Leong WF, Li L, Jia D, Ma G, Hu Y, Wang X, Chau JF, Chen YG, Mishina Y, Boast S, Yeh J, Xia L, Chen GQ, He L, Goff SP, and Baojie Li, 2012
p53 regulates neural stem cell self-renewal and differentiation via BMP-Smad1-Id1 pathway., Huijuan Liu, Deyong Jia, Jenny Chau, Lin He and Baojie Li, 2013
A critical role for p57Kip2 in the DNA damage response., Hao Jia, Jenny Fang Ling Chau, Qian Cong, and Baojie Li, 2014
Ablation of Tak1 in monocyte leads to defects in skeletal growth and bone remodeling in mice., Bing Qi, Qian Cong, Ping Li, Gang Ma, Xizhi Guo, Min Xie, Michael D. Schneider, Huijuan Liu, and Baojie Li, 2014
p53 deficiency-induced Smad1 up-regulation suppresses tumorigenesis and causes chemoresistance in colorectal cancers., Xinsen Ruan, Qiao Zuo, Jenny Chua, Hao Jia, Jinlin Lin, Xuechun Xia, Huijuan Liu, Samy L. Habib, Chuangang Fu, and Baojie Li, 2015

Stem Cells Self-Renewal Differentiation Aging Skeletal Development Bone Metabolism Osteoporosis Dna Damage Tumorigenesis Regulation Mechanisms

Huijuan Liu - Shanghai Jiao Tong University

Xuechun Xia - Shanghai Jiao Tong University

Xiaoyong Li - Shanghai Jiao Tong University

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