Professor | Zhi Qi | Asia Growth Partners

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Name	Zhi Qi
Contact Information	qizhi@xmu.edu.cn
Language ability	Chinese, English, Japanese
Province	Fujian

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Name

Zhi Qi

Contact Information

qizhi@xmu.edu.cn

Language ability

Chinese, English, Japanese

Province

Fujian

University/Lab Affiliation and Title	Xiamen University
Faculty	Medical
Academic Background & Achievements	1982-09 to 1986-07 Bachelor: Sichuan University, Applied Physics 1986-09 to 1989-07 Master: Beijing University of Technology, Applied Physics 1994-04 to 1998-03 Doctorate: Nagoya University, School of Medicine, Physiology
Work Experience	1989-09 to 1993-07 - Capital Medical University, Department of Biomedical Engineering - Assistant Lecturer, Lecturer 1998-04 to 2001-03 - Nagoya University, School of Medicine, Department of Cellular Biophysics - Research Associate 2001-04 to 2003-06 - Japan Science and Technology Agency (JST), Cellular Mechanosensing Project - Researcher 2003-06 to 2009-06 - Institute of Biophysics, Chinese Academy of Sciences/State Key Laboratory of Brain and Cognitive Science - Group Leader, Doctoral Supervisor 2009-06 to Present - Xiamen University, School of Medicine, Department of Basic Medicine - Professor, Doctoral Supervisor

University/Lab Affiliation and Title

Xiamen University

Faculty

Medical

Academic Background & Achievements

1982-09 to 1986-07 Bachelor: Sichuan University, Applied Physics
1986-09 to 1989-07 Master: Beijing University of Technology, Applied Physics
1994-04 to 1998-03 Doctorate: Nagoya University, School of Medicine, Physiology

Work Experience

1989-09 to 1993-07 - Capital Medical University, Department of Biomedical Engineering - Assistant Lecturer, Lecturer
1998-04 to 2001-03 - Nagoya University, School of Medicine, Department of Cellular Biophysics - Research Associate
2001-04 to 2003-06 - Japan Science and Technology Agency (JST), Cellular Mechanosensing Project - Researcher
2003-06 to 2009-06 - Institute of Biophysics, Chinese Academy of Sciences/State Key Laboratory of Brain and Cognitive Science - Group Leader, Doctoral Supervisor
2009-06 to Present - Xiamen University, School of Medicine, Department of Basic Medicine - Professor, Doctoral Supervisor

Areas of Focus	Ion channels and diseases, including the role of ion channels in ischemic stroke and the electrophysiological mechanisms of myocarditis development
Publications	Baifuzi reduces transient ischemic brain damage via interaction with STREX domain of BKCa channels., Chi S., Cai W., Liu P., Zhang Z., Chen X., Gao L., Qi J., Bi L., Chen L., Qi Z., 2010 Electrostatic interaction in the NH(2)-terminus accelerates inactivation of the Kv1.4 channel., Fan Z, Bi LJ, Jin G, Qi Z., 2010 Hypericin prolongs action potential duration by acting on neuronal K+ channels., Wang Y, Shi X , Qi Z ., 2010 Ion transport through dimethyl sulfoxide (DMSO) induced transient water pores in cell membranes., He F, Liu W, Zheng S, Zhou L, Ye B and Qi Z., 2012 Effect of inserting charged peptide at NH(2)-terminal on N-type inactivation of Kv1.4 channel., Fan Z, Zhang Z, Fu M, Qi Z, Xiao Z., 2013 Structure-activity relationship of Baifuzi-cerebrosides on BKCa channel activation., Zhou L.#, Zhang Y#., Gao L., Ye Y., Qi J., Qi Z., 2014 Palmitoylation of STREX domain confers cerebroside sensitivity to the BKCa channel., Zhang Y., Zhou L., Zou J., Wang M., Qi J., Qi Z., 2014 Chlorpromazine confers neuroprotection against brain ischemia by activating BKCa channel., Hua-Juan Li, Yu-Jiao Zhang, Li Zhou, Feng Han, Ming-Yan Wang, Mao-Qiang Xue, Zhi Qi., 2014 Cholesterol modulates function of connexin 43 gap junction channel via PKC pathway in H9c2 cells., Jun Zou, Xiao-Yang Yue, Sheng-Chao Zheng, Guanwei Zhang, He Chang, Yan-Chun Liao, Ye Zhang, Mao-Qiang Xue, Zhi Qi, 2014 Nitric oxide inhibits the basolateral 10-pS Cl- channels through cGMP/PKG signaling pathway in the thick ascending limb of C57BL/6 mice., Wu P, Gao Z, Ye S, Qi Z*., 2016

Areas of Focus

Ion channels and diseases, including the role of ion channels in ischemic stroke and the electrophysiological mechanisms of myocarditis development

Publications

Baifuzi reduces transient ischemic brain damage via interaction with STREX domain of BKCa channels., Chi S., Cai W., Liu P., Zhang Z., Chen X., Gao L., Qi J., Bi L., Chen L*., Qi Z*., 2010
Electrostatic interaction in the NH(2)-terminus accelerates inactivation of the Kv1.4 channel., Fan Z, Bi LJ*, Jin G*, Qi Z*., 2010
Hypericin prolongs action potential duration by acting on neuronal K+ channels., Wang Y, Shi X *, Qi Z *., 2010
Ion transport through dimethyl sulfoxide (DMSO) induced transient water pores in cell membranes., He F, Liu W, Zheng S, Zhou L, Ye B* and Qi Z*., 2012
Effect of inserting charged peptide at NH(2)-terminal on N-type inactivation of Kv1.4 channel., Fan Z, Zhang Z, Fu M, Qi Z*, Xiao Z*., 2013
Structure-activity relationship of Baifuzi-cerebrosides on BKCa channel activation., Zhou L.#, Zhang Y#., Gao L., Ye Y., Qi J*., Qi Z*., 2014
Palmitoylation of STREX domain confers cerebroside sensitivity to the BKCa channel., Zhang Y., Zhou L., Zou J., Wang M., Qi J., Qi Z*., 2014
Chlorpromazine confers neuroprotection against brain ischemia by activating BKCa channel., Hua-Juan Li, Yu-Jiao Zhang, Li Zhou, Feng Han, Ming-Yan Wang, Mao-Qiang Xue*, Zhi Qi*., 2014
Cholesterol modulates function of connexin 43 gap junction channel via PKC pathway in H9c2 cells., Jun Zou*, Xiao-Yang Yue, Sheng-Chao Zheng, Guanwei Zhang, He Chang, Yan-Chun Liao, Ye Zhang, Mao-Qiang Xue, Zhi Qi*, 2014
Nitric oxide inhibits the basolateral 10-pS Cl- channels through cGMP/PKG signaling pathway in the thick ascending limb of C57BL/6 mice., Wu P, Gao Z, Ye S, Qi Z*., 2016

Ion Channels Diseases Ischemic Stroke Electrophysiology Myocarditis Development Mechanisms Role Research Focus

Zhengping Xu - Zhejiang University

Yan Luo - Zhejiang University

Shumin Duan - Zhejiang University

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