Professor | Yingjun Zhao | Asia Growth Partners

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Name	Yingjun Zhao
Contact Information	yjzhao@xmu.edu.cn
Language ability	Chinese, English
Province	Fujian

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Name

Yingjun Zhao

Contact Information

yjzhao@xmu.edu.cn

Language ability

Chinese, English

Province

Fujian

University/Lab Affiliation and Title	Xiamen University
Faculty	Medical
Academic Background & Achievements	2003-2007 Bachelor: Xiamen University, School of Life Sciences 2007-2009 Master: Xiamen University, Institute of Biomedical Sciences 2009-2012 PhD: Xiamen University, School of Pharmacy and Sanford Burnham Prebys Medical Discovery Institute Achievement: Published 40 SCI papers, with over 2200 citations in the last 5 years
Work Experience	2012-2018 Postdoctoral Researcher and Research Assistant Professor: Sanford Burnham Prebys Medical Discovery Institute, USA 2018-present Professor: Xiamen University, School of Medicine
Awards	2020: National High-level Young Talent 2018: Minjiang Scholar Distinguished Professor 2017: Joy Cappel Young Investigator Award, USA

University/Lab Affiliation and Title

Xiamen University

Faculty

Medical

Academic Background & Achievements

2003-2007 Bachelor: Xiamen University, School of Life Sciences
2007-2009 Master: Xiamen University, Institute of Biomedical Sciences
2009-2012 PhD: Xiamen University, School of Pharmacy and Sanford Burnham Prebys Medical Discovery Institute
Achievement: Published 40 SCI papers, with over 2200 citations in the last 5 years

Work Experience

2012-2018 Postdoctoral Researcher and Research Assistant Professor: Sanford Burnham Prebys Medical Discovery Institute, USA
2018-present Professor: Xiamen University, School of Medicine

Awards

2020: National High-level Young Talent
2018: Minjiang Scholar Distinguished Professor
2017: Joy Cappel Young Investigator Award, USA

Areas of Focus	Neurodegenerative Diseases Pathophysiology
Publications	P-tau217 correlates with neurodegeneration in Alzheimer's disease, and targeting p-tau217 with immunotherapy ameliorates murine tauopathy., Zhang D, Zhang W, Ming C, Gao X, Yuan H, Lin X, Mao X, Wang C, Guo X, Du Y, Shao L, Yang R, Lin Z, Wu X, Huang TY, Wang Z, Zhang YW, Xu H, Zhao Y., 2024 Passive immunotherapy for Alzheimer's disease., Guo X, Yan L, Zhang D, Zhao Y., 2024 Simplified regimen of combined low-dose rituximab for autoimmune encephalitis with neuronal surface antibodies., Du Y, Zhao C, Liu J, Li C, Yan Q, Li L, Hao Y, Yao D, Si H, Zhao Y, Zhang W., 2022 Downregulating PTBP1 fails to convert astrocytes into hippocampal neurons and to alleviate symptoms in Alzheimer's mouse models., Guo T#, Pan X#, Jiang G, Zhang D, Qi J, Shao L, Wang Z, Xu H, Zhao Y., 2022 APOE interacts with ACE2 inhibiting SARS-CoV-2 cellular entry and inflammation in COVID-19 patients., Zhang H#, Shao L#, Lin Z#, Long QX#, Yuan H#, Cai L, Jiang G, Guo X, Yang R, Zhang Z, Zhang B, Liu F, Li Z, Ma Q, Zhang YW, Huang AL, Wang Z, Zhao Y, Xu H., 2022 Molecular and cellular mechanisms underlying the pathogenesis of Alzheimer's disease., Guo T#, Zhang D#, Zeng Y, Huang TY, Xu H, Zhao Y., 2020 Appoptosin-Mediated Caspase Cleavage of Tau Contributes to Progressive Supranuclear Palsy Pathogenesis., Zhao Y, Tseng IC, Heyser CJ, Rockenstein E, Mante M, Adame A, Zheng Q, Huang T, Wang X, Arslan PE, Chakrabarty P, Wu C, Bu G, Mobley WC, Zhang YW, St George- Hyslop P, Masliah E, Fraser P, Xu H., 2015 TREM2 is a receptor for β-amyloid which mediates microglial function., Zhao Y#, Wu X#, Li X#, Jiang LL#, Gui X, Liu Y, Sun Y, Zhu B, Pina-Crespo JC, Zhang M, Zhang N, Chen X, Bu G, An Z, Huang TY, Xu H., 2018 Inhibition of PKCδ reduces amyloid-β levels and reverses Alzheimer disease phenotypes., Du Y#, Zhao Y#, Li C#, Zheng Q, Tian J, Li Z, Huang T, Zhang W, Xu H., 2018 SORLA attenuates EphA4 signaling and β-amyloid-induced neurodegeneration., Huang T#, Zhao Y#, Jiang LL, Li X, Liu Y, Sun Y, Pina-Crespo JC, Zhu B, Masliah E, Willnow TE, Pasquale EB, Xu H., 2017 Intracellular-trafficking of TREM2 is regulated by presenilin 1., Zhao Y#, Li X#, Huang T, Jiang LL, Zhang M, Cheng I HJ, Wang X, Bu G, Zhang YW, Wang Q, Xu H*., 2017 CutA divalent cation tolerance homolog (E. coli) (CUTA) regulates β-cleavage of β-amyloid precursor protein (APP) through interacting with β-Site APP cleaving protein 1 (BACE1)., Zhao Y, Wang Y, Hu J, Zhang X, Zhang YW., 2012

Areas of Focus

Neurodegenerative Diseases
Pathophysiology

Publications

P-tau217 correlates with neurodegeneration in Alzheimer's disease, and targeting p-tau217 with immunotherapy ameliorates murine tauopathy., Zhang D, Zhang W, Ming C, Gao X, Yuan H, Lin X, Mao X, Wang C, Guo X, Du Y, Shao L, Yang R, Lin Z, Wu X, Huang TY, Wang Z, Zhang YW, Xu H, Zhao Y*., 2024
Passive immunotherapy for Alzheimer's disease., Guo X, Yan L, Zhang D, Zhao Y*., 2024
Simplified regimen of combined low-dose rituximab for autoimmune encephalitis with neuronal surface antibodies., Du Y, Zhao C, Liu J, Li C, Yan Q, Li L, Hao Y, Yao D, Si H, Zhao Y*, Zhang W*., 2022
Downregulating PTBP1 fails to convert astrocytes into hippocampal neurons and to alleviate symptoms in Alzheimer's mouse models., Guo T#, Pan X#, Jiang G, Zhang D, Qi J, Shao L, Wang Z, Xu H*, Zhao Y*., 2022
APOE interacts with ACE2 inhibiting SARS-CoV-2 cellular entry and inflammation in COVID-19 patients., Zhang H#, Shao L#, Lin Z#, Long QX#, Yuan H#, Cai L, Jiang G, Guo X, Yang R, Zhang Z, Zhang B, Liu F, Li Z, Ma Q, Zhang YW, Huang AL*, Wang Z*, Zhao Y*, Xu H*., 2022
Molecular and cellular mechanisms underlying the pathogenesis of Alzheimer's disease., Guo T#, Zhang D#, Zeng Y, Huang TY*, Xu H*, Zhao Y*., 2020
Appoptosin-Mediated Caspase Cleavage of Tau Contributes to Progressive Supranuclear Palsy Pathogenesis., Zhao Y, Tseng IC, Heyser CJ, Rockenstein E, Mante M, Adame A, Zheng Q, Huang T, Wang X, Arslan PE, Chakrabarty P, Wu C, Bu G, Mobley WC, Zhang YW, St George- Hyslop P, Masliah E, Fraser P, Xu H., 2015
TREM2 is a receptor for β-amyloid which mediates microglial function., Zhao Y#, Wu X#, Li X#, Jiang LL#, Gui X, Liu Y, Sun Y, Zhu B, Pina-Crespo JC, Zhang M, Zhang N, Chen X, Bu G, An Z, Huang TY, Xu H., 2018
Inhibition of PKCδ reduces amyloid-β levels and reverses Alzheimer disease phenotypes., Du Y#, Zhao Y#, Li C#, Zheng Q, Tian J, Li Z, Huang T, Zhang W, Xu H., 2018
SORLA attenuates EphA4 signaling and β-amyloid-induced neurodegeneration., Huang T#, Zhao Y#, Jiang LL, Li X, Liu Y, Sun Y, Pina-Crespo JC, Zhu B, Masliah E, Willnow TE, Pasquale EB, Xu H., 2017
Intracellular-trafficking of TREM2 is regulated by presenilin 1., Zhao Y#, Li X#, Huang T, Jiang LL, Zhang M, Cheng I HJ, Wang X, Bu G, Zhang YW, Wang Q*, Xu H*., 2017
CutA divalent cation tolerance homolog (E. coli) (CUTA) regulates β-cleavage of β-amyloid precursor protein (APP) through interacting with β-Site APP cleaving protein 1 (BACE1)., Zhao Y, Wang Y, Hu J, Zhang X, Zhang YW., 2012

Alzheimer'S Disease Neurodegeneration Immunotherapy Genetic Variation Tau Protein Microglia Trem2 Sorla Aβ Toxicity Synaptic Toxicity

Fang Marong - Zhejiang University

Xiaofen Chen - Xiamen University

Xin Wang - Xiamen University

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