Professor | Liang Ge | Asia Growth Partners

Name	Liang Ge
Contact Information	liangge@mail.tsinghua.edu.cn
Language ability	English, Chinese
Province	Beijing

Name

Liang Ge

Contact Information

liangge@mail.tsinghua.edu.cn

Language ability

English, Chinese

Province

Beijing

University/Lab Affiliation and Title	Tsinghua University
Faculty	Life Sciences
Academic Background & Achievements	2001-2005 Bachelor: Shandong Normal University, School of Life Sciences 2005-2011 PhD in Biochemistry: Shanghai Institute for Biological Sciences, CAS Postdoctoral Researcher at University of California, Berkeley (2011-2015)
Work Experience	2015-2017 - University of California, Berkeley - Research Specialist 2017-present - Tsinghua University, School of Life Sciences - Associate Professor
Awards	NIH Pathway to Independence Award (K99/R00) (2015-2017) Jane Coffin Child Foundation Fellowship (2013-2015) Human Frontier Science Program Fellowship (2012)

University/Lab Affiliation and Title

Tsinghua University

Faculty

Life Sciences

Academic Background & Achievements

2001-2005 Bachelor: Shandong Normal University, School of Life Sciences
2005-2011 PhD in Biochemistry: Shanghai Institute for Biological Sciences, CAS
Postdoctoral Researcher at University of California, Berkeley (2011-2015)

Work Experience

2015-2017 - University of California, Berkeley - Research Specialist
2017-present - Tsinghua University, School of Life Sciences - Associate Professor

Awards

NIH Pathway to Independence Award (K99/R00) (2015-2017)
Jane Coffin Child Foundation Fellowship (2013-2015)
Human Frontier Science Program Fellowship (2012)

Areas of Focus	Cellular autophagy Non-classical secretion Organelle interaction
Publications	CCT2 is an aggrephagy receptor for clearance of solid protein aggregates, Liang Ge et al., 2022 A new type of ERGIC-ERES membrane contact mediated by TMED9 and SEC12 is required for autophagosome biogenesis, Liang Ge et al., 2022 A translocation pathway for vesicle-mediated unconventional protein secretion, Liang Ge et al., 2020 Remodeling of ER-exit sites initiates a membrane supply pathway for autophagosome biogenesis, Liang Ge et al., 2017 Translocation of interleukin-1β into a vesicle intermediate in autophagy-mediated secretion, Liang Ge et al., 2015 Phosphatidylinositol 3-kinase and COPII generate LC3 lipidation vesicles from the ER-Golgi intermediate compartment, Liang Ge et al., 2014 The ER-Golgi intermediate compartment is a key membrane source for the LC3 lipidation step of autophagosome biogenesis, Liang Ge et al., 2013

Areas of Focus

Cellular autophagy
Non-classical secretion
Organelle interaction

Publications

CCT2 is an aggrephagy receptor for clearance of solid protein aggregates, Liang Ge et al., 2022
A new type of ERGIC-ERES membrane contact mediated by TMED9 and SEC12 is required for autophagosome biogenesis, Liang Ge et al., 2022
A translocation pathway for vesicle-mediated unconventional protein secretion, Liang Ge et al., 2020
Remodeling of ER-exit sites initiates a membrane supply pathway for autophagosome biogenesis, Liang Ge et al., 2017
Translocation of interleukin-1β into a vesicle intermediate in autophagy-mediated secretion, Liang Ge et al., 2015
Phosphatidylinositol 3-kinase and COPII generate LC3 lipidation vesicles from the ER-Golgi intermediate compartment, Liang Ge et al., 2014
The ER-Golgi intermediate compartment is a key membrane source for the LC3 lipidation step of autophagosome biogenesis, Liang Ge et al., 2013

Autophagy Stress Response Cellular Cleaning Molecular Mechanisms Biological Significance Human Diseases Cell Biology Biochemistry Animal Models Computational Biology

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Yeguang Chen - Tsinghua University

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