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Lei Wei
leiwei@whu.edu.cn
Chinese, English, French
Hubei
Wuhan University
Basic Medical Sciences
  • 2001-2004: PhD in Bioengineering, University of Nancy, France
  • 2000-2003: PhD in Cardiovascular Diseases, Wuhan University, China
  • 1994-1997: Master's in Pathophysiology, Hubei Medical University, China
  • 2013-Present: Party Secretary, Vice Director, Department of Pathophysiology, Wuhan University
  • 2010-2014: Vice Director, Wuhan University Center for Structural Biology
  • 2006-Present: Professor, Supervisor for PhD, Department of Pathophysiology, Wuhan University
  • 2001-2006: Associate Professor, Supervisor for Masters, Department of Pathophysiology, Wuhan University
  • 1998-2001: Lecturer, Department of Pathophysiology, Wuhan University
Tumor molecular pathology and pathophysiology
Cardiovascular pathophysiology
  • CircRNA inhibits DNA damage repair by interacting with host gene, Wei Lei, 2020
  • The Wnt/beta-catenin/VASP positive feedback loop drives cell proliferation and migration in breast cancer, Wei Lei, 2020
  • MAGI2‐AS3 inhibits breast cancer by downregulating DNA methylation of MAGI2, Wei Lei, 2021
  • Histone demethylase KDM4B promotes DNA damage by activating longinterspersed nuclear element-1 (LINE-1), Wei Lei, 2019
  • CREB1/Lin28/miR-638/VASP interactive network drives the development of breast cancer, Wei Lei, 2019
  • Chlorotoxin targets ERalpha/VASP signaling pathway to combat breast cancer, Wei Lei, 2019
  • Tumor promoter TPA activates Wnt/β-catenin signaling in a casein kinase 1-dependent manner, Wei Lei, 2018
  • CSCD: a database for cancer-specific circular RNAs, Wei Lei, 2017
  • Prodigiosin inhibits Wnt/β-catenin signaling and exerts anticancer activity in breast cancer cells, Wei Lei, 2018
  • MicroRNA-610 inhibits the migration and invasion of gastric cancer cells by suppressing the expression of vasodilator-stimulated phosphoprotein, Wei Lei, 2012
Tumor Molecular Pathology Pathophysiology Cardiovascular Research Clinical Translational Medicine Cell Regulation Molecular Targets

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