Areas of Focus
- Molecular, cellular, and genetic techniques to study growth factors and receptors
- Structural biology, cell signaling, neural development, metabolic regulation
Work Experience
- 2020 - Peking University, McGovern Institute for Brain Research and Beijing Brain Science and Brain-Inspired Research Center - Established new research group
- 2012 - National University of Singapore, Yong Loo Lin School of Medicine, Department of Physiology - Joint appointment as Professor
- 2004 - Member of the Nobel Assembly at Karolinska Institute
- 1996 - Karolinska Institute, Department of Neuroscience - Professor of Neuroscience
- Postdoctoral research under Håkan Persson at Karolinska Institute
Academic Background & Achievements
- Studied Biology at University of Buenos Aires
- PhD work under Carlos Frasch at Leloir Institute
Publications
- Activin receptor ALK4 promotes adipose tissue hyperplasia by suppressing differentiation of adipocyte precursors, Lee ES, Guo T, Srivastava RK, Shabbir A, Ibáñez CF, 2023
- Convergent dopamine and ALK4 signaling to PCBP1 controls FosB alternative splicing and cocaine behavioral sensitization, Krapacher FA, Fernández-Suárez D, Andersson A, Carrier-Ruiz A, Ibáñez CF, 2022
- Adult medial habenula neurons require GDNF receptor GFRα1 for synaptic stability and function, Fernández-Suárez D, Krapacher FA, Pietrajtis K, Andersson A, Kisiswa L, Carrier-Ruiz A, Diana MA, Ibáñez CF, 2021
- Inactive variants of death receptor p75(NTR) reduce Alzheimer`s neuropathology by interfering with APP internalization, Yi, C., Goh, K. Y., Wong, L. W., Ramanujan, A., Tanaka, K., Sajikumar, S., & Ibanez, C. F., 2021
- ALK4 coordinates extracellular and intrinsic signals to regulate development of cortical somatostatin interneurons, Gongrich, C., Krapacher, F. A., Munguba, H., Fernandez-Suarez, D., Andersson, A., Hjerling-Leffler, J., & Ibanez, C. F., 2020
- Control of brown adipose tissue adaptation to nutrient stress by the activin receptor ALK7, Marmol, P., Krapacher, F., & Ibanez, C. F., 2020
- Adipocyte ALK7 links nutrient overload to catecholamine resistance in obesity, Guo, T., Marmol, P., Moliner, A., Bjornholm, M., Zhang, C., Shokat, K. M., & Ibanez, C. F., 2014
- Activation of the p75 neurotrophin receptor through conformational rearrangement of disulphide-linked receptor dimers, Vilar, M., Charalampopoulos, I., Kenchappa, R. S., Simi, A., Karaca, E., Reversi, A., . . . Ibanez, C. F., 2009
- Activin B receptor ALK7 is a negative regulator of pancreatic beta-cell function, Bertolino, P., Holmberg, R., Reissmann, E., Andersson, O., Berggren, P. O., & Ibanez, C. F., 2008
- GDNF and GFRalpha1 promote formation of neuronal synapses by ligand-induced cell adhesion, Ledda, F., Paratcha, G., Sandoval-Guzman, T., & Ibanez, C. F., 2007
- Bex1, a novel interactor of the p75 neurotrophin receptor, links neurotrophin signaling to the cell cycle, Vilar, M., Murillo-Carretero, M., Mira, H., Magnusson, K., Besset, V., & Ibanez, C. F., 2006
- GDNF and GFRalpha1 promote differentiation and tangential migration of cortical GABAergic neurons, Pozas, E., & Ibanez, C. F., 2005
- The neural cell adhesion molecule NCAM is an alternative signaling receptor for GDNF family ligands, Paratcha, G., Ledda, F., & Ibanez, C. F., 2003
- Target-derived GFRalpha1 as an attractive guidance signal for developing sensory and sympathetic axons via activation of Cdk5, Ledda, F., Paratcha, G., & Ibanez, C. F., 2002