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Zhang Peng
Medical School
Wuhan University
Hubei
Language: Chinese, English
Nafld
Hcc
Pathogenesis
Animal Models
Rna-Seq
Proteomics
Molecular Biology
Therapeutic Targets
Prevention
Mechanism
Areas of Focus
- Non-alcoholic fatty liver disease and hepatocellular carcinoma pathogenesis
Work Experience
- 2015.07-2017.07 Postdoctoral Fellow, School of Basic Medical Sciences, Wuhan University
- 2017.08-2020.08 Assistant Research Fellow, Zhongnan Hospital of Wuhan University
- 2020.09-present Distinguished Research Fellow, School of Basic Medical Sciences, Wuhan University
Academic Background & Achievements
- 2002.09-2006.06 Bachelor of Biological Sciences, Jiamusi University
- 2007.09-2010.06 Master of Zoology, Lanzhou University
- 2010.09-2014.06 PhD in Genetics, Shanghai Jiao Tong University
- Has led 4 national and provincial level projects
Publications
- Cardiolipin Synthase 1 Ameliorates NASH Through Activating Transcription Factor 3 Transcriptional Inactivation, Zhang P*, Mei Z*, 2020
- TNFAIP3 Interacting Protein 3 Overexpression Suppresses Nonalcoholic Steatohepatitis by Blocking TAK1 Activation, Zhang P*, Xu H*, She ZG*, Li H*, 2020
- Association of Inpatient Use of Angiotensin-Converting Enzyme Inhibitors and Angiotensin II Receptor Blockers with Mortality Among Patients With Hypertension Hospitalized With COVID-19, Zhang P*, Liu PP*, Li H*, 2020
- Hepatic Interferon Regulatory Factor 6 Alleviates Liver Steatosis and Metabolic Disorder by Transcriptionally Suppressing Peroxisome Proliferator-Activated Receptor γ in Mice, Zhang P*, Li H*, 2019
- Tumor progression locus 2 in hepatocytes potentiates both liver and systemic metabolic disorders in mice, Zhang P*, She ZG*, 2019
- Tmbim1 is a multivesicular body regulator that protects against non-alcoholic fatty liver disease in mice and monkeys by targeting the lysosomal degradation of Tlr4, Zhang P*, Li H*, 2017
- Smad4 Deficiency in Smooth Muscle Cells Initiates the Formation of Aortic Aneurysm, Zhang P*, Yang X*, 2016
- The ubiquitin E3 ligase TRAF6 exacerbates pathological cardiac hypertrophy via TAK1-dependent signaling, Zhang P*, Li H*, 2016
