Professor | Hongbo Weng | Asia Growth Partners

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Name	Hongbo Weng
Contact Information	hbweng@fudan.edu.cn
Language ability	Chinese, English
Province	Shanghai

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Name

Hongbo Weng

Contact Information

hbweng@fudan.edu.cn

Language ability

Chinese, English

Province

Shanghai

University/Lab Affiliation and Title	Fudan University
Faculty	Pharmaceutical Sciences
Academic Background & Achievements	2004-2008 PhD in Pharmacology: Fudan University 1994-1997 Master's in Pharmacology: Shenyang Pharmaceutical University 1990-1994 Bachelor's in Pharmaceutical Preparation: Shenyang Pharmaceutical University
Work Experience	2009-2011 - University of Maryland School of Medicine - Postdoctoral Researcher 2000-Present - Fudan University School of Pharmacy - Lecturer 1997-2000 - Shanghai Medical University School of Pharmacy - Teaching Assistant 1994-1997 - Shenyang Pharmaceutical University - Master's Student

University/Lab Affiliation and Title

Fudan University

Faculty

Pharmaceutical Sciences

Academic Background & Achievements

2004-2008 PhD in Pharmacology: Fudan University
1994-1997 Master's in Pharmacology: Shenyang Pharmaceutical University
1990-1994 Bachelor's in Pharmaceutical Preparation: Shenyang Pharmaceutical University

Work Experience

2009-2011 - University of Maryland School of Medicine - Postdoctoral Researcher
2000-Present - Fudan University School of Pharmacy - Lecturer
1997-2000 - Shanghai Medical University School of Pharmacy - Teaching Assistant
1994-1997 - Shenyang Pharmaceutical University - Master's Student

Areas of Focus	Research on Anti-Diabetic Drugs
Publications	Oxidative stress-induced JNK1/2 activation triggers pro-apoptotic signaling and apoptosis that leads to diabetic embryopathy, Xuezheng Li, Hongbo Weng, Cheng Xu, E. Albert Reece, Peixin Yang, 2012 SOD1 suppresses maternal hyperglycemia-increased iNOS expression and consequent nitrosative stress in diabetic embryopathy, Weng H, Li X, Reece EA, Yang P, 2012 Increased secretion and expression of amylin in spontaneously diabetic Goto-Kakizaki rats treated with rhGLP-1 (7–36), Weng H.B., Gu Q., Liu M., Cheng N.N., Li D., Gao X., 2008 SOD1 overexpression in vivo blocks hyperglycemia-induced specific PKC isoforms: substrate activation and consequent lipid peroxidation in diabetic embryopathy, Xuezheng Li, Hongbo Weng, E. Albert Reece, Peixin Yang, 2011 Trehalose prevents neural tube defects by correcting maternal diabetes-suppressed autophagy and neurogenesis, Cheng Xu, Xuezheng Li, Fang Wang, Hongbo Weng, 2013 佛司可林类化合物对离体豚鼠气管条舒张作用的研究, 汪亚勤，曹水娟，殷嘉珺，黄建明，翁鸿博, 2013 葛根黄豆甙元对沙土鼠脑缺血及再灌注损伤的神经保护作用(英文), 翁鸿博，刘玉兰，程能能, 2012 Qi盐对肾性和自发性高血压大鼠血压的影响(英文), 翁鸿博, 张一鸣，李端，程能能, 2005 Qi盐对正常及糖尿病模型大鼠血糖的影响(英文), 翁鸿博, 张一鸣，李端，程能能, 2004 葛根黄豆苷元对脑缺血的保护作用及机制探讨, 翁鸿博，马涛，刘玉兰, 1999 葛根黄豆苷元的抗缺氧缺血作用, 翁鸿博，马涛，尤越人, 1999

Areas of Focus

Research on Anti-Diabetic Drugs

Publications

Oxidative stress-induced JNK1/2 activation triggers pro-apoptotic signaling and apoptosis that leads to diabetic embryopathy, Xuezheng Li, Hongbo Weng, Cheng Xu, E. Albert Reece, Peixin Yang, 2012
SOD1 suppresses maternal hyperglycemia-increased iNOS expression and consequent nitrosative stress in diabetic embryopathy, Weng H, Li X, Reece EA, Yang P, 2012
Increased secretion and expression of amylin in spontaneously diabetic Goto-Kakizaki rats treated with rhGLP-1 (7–36), Weng H.B., Gu Q., Liu M., Cheng N.N., Li D., Gao X., 2008
SOD1 overexpression in vivo blocks hyperglycemia-induced specific PKC isoforms: substrate activation and consequent lipid peroxidation in diabetic embryopathy, Xuezheng Li, Hongbo Weng, E. Albert Reece, Peixin Yang, 2011
Trehalose prevents neural tube defects by correcting maternal diabetes-suppressed autophagy and neurogenesis, Cheng Xu, Xuezheng Li, Fang Wang, Hongbo Weng, 2013
佛司可林类化合物对离体豚鼠气管条舒张作用的研究, 汪亚勤，曹水娟，殷嘉珺，黄建明，翁鸿博, 2013
葛根黄豆甙元对沙土鼠脑缺血及再灌注损伤的神经保护作用(英文), 翁鸿博，刘玉兰，程能能, 2012
Qi盐对肾性和自发性高血压大鼠血压的影响(英文), 翁鸿博, 张一鸣，李端，程能能, 2005
Qi盐对正常及糖尿病模型大鼠血糖的影响(英文), 翁鸿博, 张一鸣，李端，程能能, 2004
葛根黄豆苷元对脑缺血的保护作用及机制探讨, 翁鸿博，马涛，刘玉兰, 1999
葛根黄豆苷元的抗缺氧缺血作用, 翁鸿博，马涛，尤越人, 1999

Diabetes Pharmacology Drug Research Anti-Diabetic Mechanism Study Complement Inhibitors Lupus Molecular Biology Immunology Hypertension

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