Professor | Boxun Lu | Asia Growth Partners

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Name

Boxun Lu

Contact Information

luboxun@fudan.edu.cn

Language ability

Chinese, English

Province

Shanghai

University/Lab Affiliation and Title	Fudan University
Faculty	Life Sciences
Academic Background & Achievements	Bachelor's Degree: Fudan University Ph.D.: University of Pennsylvania National Distinguished Youth Fund National Excellent Youth Fund National Original Project Fund Newton Advanced Fellowship from the British Academy of Medical Sciences Xplorer Prize Ministry of Education Youth Science Award Wu Rui Memorial Foundation Gu Xiaocheng Award Shanghai Youth Science and Technology Talent
Awards	Xplorer Prize Ministry of Education Youth Science Award Wu Rui Memorial Foundation Gu Xiaocheng Award Shanghai Youth Science and Technology Talent

University/Lab Affiliation and Title

Fudan University

Faculty

Life Sciences

Academic Background & Achievements

Awards

Areas of Focus	Neurodegenerative Diseases Original Drug Development Strategies
Publications	Allele-selective lowering of mutant HTT protein by HTT-LC3 linker compounds, Li Z, et.al, Ding Y, Fei Y, Lu B*, 2019 Targeting Gpr52 lowers mutant HTT levels and rescues Huntington’s disease-associated phenotypes, Song H, et.al, Lu B, 2018 Suppression of MAPK11 or HIPK3 reduces mutant Huntingtin levels in Huntington’s disease models, Yu M, Fu Y, Liang Y, et.al, Lu B, 2017 A Toxic Mutant Huntingtin Species Is Resistant to Selective Autophagy, Fu Y, Wu P, Pan Y, Sun X, Difiglia M, Lu B, 2017 A Striatal-enriched Intronic GPCR Modulates Huntingtin Levels and Toxicity, Yao Y, Cui X, Al-Ramah I, Sun X, Li B, Hou J, Difiglia M, Palacino J, Wu Z, Ma L, Botas J, Boxun Lu, 2015

Areas of Focus

Neurodegenerative Diseases
Original Drug Development Strategies

Publications

Allele-selective lowering of mutant HTT protein by HTT-LC3 linker compounds, Li Z, et.al, Ding Y*, Fei Y*, Lu B*, 2019
Targeting Gpr52 lowers mutant HTT levels and rescues Huntington’s disease-associated phenotypes, Song H, et.al, Lu B, 2018
Suppression of MAPK11 or HIPK3 reduces mutant Huntingtin levels in Huntington’s disease models, Yu M, Fu Y, Liang Y, et.al, Lu B, 2017
A Toxic Mutant Huntingtin Species Is Resistant to Selective Autophagy, Fu Y, Wu P, Pan Y, Sun X, Difiglia M, Lu B, 2017
A Striatal-enriched Intronic GPCR Modulates Huntingtin Levels and Toxicity, Yao Y, Cui X, Al-Ramah I, Sun X, Li B, Hou J, Difiglia M, Palacino J, Wu Z, Ma L, Botas J, Boxun Lu, 2015

Neurodegenerative Diseases Drug Development Biophysics Chemical Biology Huntington'S Disease Htt Protein Gpr52 Mapk11 Hipk3 Autophagy

Jing Liu - Central South University

Yan Zhao - Southern University of Science and Technology

Hong Zhang - University of Chinese Academy of Sciences

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