Professor | Boxun Lu | Asia Growth Partners

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Name

Boxun Lu

Contact Information

luboxun@fudan.edu.cn

Language ability

Chinese, English

Province

Shanghai

University/Lab Affiliation and Title	Fudan University
Faculty	Life Sciences
Academic Background & Achievements	Graduated with a Bachelor's degree from Fudan University Graduated with a Ph.D. from the University of Pennsylvania Received National Outstanding Youth Fund Received National Excellent Youth Fund Received National Original Project Fund Received Newton Advanced Fellowship from the British Academy of Medical Sciences Received Xplorer Prize Received Youth Science Award from the Ministry of Education Received Gu Xiaocheng Award from the Wu Rui Memorial Foundation Received Shanghai Youth Science and Technology Talent Award
Awards	National Outstanding Youth Fund National Excellent Youth Fund National Original Project Fund Newton Advanced Fellowship from the British Academy of Medical Sciences Xplorer Prize Youth Science Award from the Ministry of Education Gu Xiaocheng Award from the Wu Rui Memorial Foundation Shanghai Youth Science and Technology Talent Award

University/Lab Affiliation and Title

Fudan University

Faculty

Life Sciences

Academic Background & Achievements

Graduated with a Bachelor's degree from Fudan University
Graduated with a Ph.D. from the University of Pennsylvania
Received National Outstanding Youth Fund
Received National Excellent Youth Fund
Received National Original Project Fund
Received Newton Advanced Fellowship from the British Academy of Medical Sciences
Received Xplorer Prize
Received Youth Science Award from the Ministry of Education
Received Gu Xiaocheng Award from the Wu Rui Memorial Foundation
Received Shanghai Youth Science and Technology Talent Award

Awards

Areas of Focus	Neurodegenerative Diseases Original Drug Development Strategies
Publications	Allele-selective lowering of mutant HTT protein by HTT-LC3 linker compounds, Li Z, et.al, Ding Y, Fei Y, Lu B*, 2019 Targeting Gpr52 lowers mutant HTT levels and rescues Huntington’s disease-associated phenotypes, Song H, et.al, Lu B, 2018 Suppression of MAPK11 or HIPK3 reduces mutant Huntingtin levels in Huntington’s disease models, Yu M, Fu Y, Liang Y, et.al, Lu B, 2017 A Toxic Mutant Huntingtin Species Is Resistant to Selective Autophagy, Fu Y, Wu P, Pan Y, Sun X, Difiglia M, Lu B, 2017 A Striatal-enriched Intronic GPCR Modulates Huntingtin Levels and Toxicity, Yao Y, Cui X, Al-Ramah I, Sun X, Li B, Hou J, Difiglia M, Palacino J, Wu Z, Ma L, Botas J, Boxun Lu, 2015

Areas of Focus

Neurodegenerative Diseases
Original Drug Development Strategies

Publications

Allele-selective lowering of mutant HTT protein by HTT-LC3 linker compounds, Li Z, et.al, Ding Y*, Fei Y*, Lu B*, 2019
Targeting Gpr52 lowers mutant HTT levels and rescues Huntington’s disease-associated phenotypes, Song H, et.al, Lu B, 2018
Suppression of MAPK11 or HIPK3 reduces mutant Huntingtin levels in Huntington’s disease models, Yu M, Fu Y, Liang Y, et.al, Lu B, 2017
A Toxic Mutant Huntingtin Species Is Resistant to Selective Autophagy, Fu Y, Wu P, Pan Y, Sun X, Difiglia M, Lu B, 2017
A Striatal-enriched Intronic GPCR Modulates Huntingtin Levels and Toxicity, Yao Y, Cui X, Al-Ramah I, Sun X, Li B, Hou J, Difiglia M, Palacino J, Wu Z, Ma L, Botas J, Boxun Lu, 2015

Neurodegenerative Diseases Drug Development Biophysics Chemical Biology Huntington'S Disease Autophagy Mutant Htt Gpr52 Mapk11 Hipk3

Jing Liu - Central South University

Yan Zhao - Southern University of Science and Technology

Hong Zhang - University of Chinese Academy of Sciences

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