Professor | Renhong Du | Asia Growth Partners

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Name	Renhong Du
Contact Information	drh@njmu.edu.cn
Language ability	Chinese, English
Province	Jiangsu

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Name

Renhong Du

Contact Information

drh@njmu.edu.cn

Language ability

Chinese, English

Province

Jiangsu

University/Lab Affiliation and Title	Nanjing Medical University
Faculty	Basic Medical Sciences
Academic Background & Achievements	1998-2003 Clinical Medicine: Wannan Medical College 2005-2010 PhD in Pharmacology: Sun Yat-sen University Published 16 SCI papers as first/corresponding author with a cumulative impact factor of 80
Work Experience	2010-2018 - Nanjing Medical University - Lecturer in Pharmacology 2018-present - Nanjing Medical University - Associate Professor in Pharmacology

University/Lab Affiliation and Title

Nanjing Medical University

Faculty

Basic Medical Sciences

Academic Background & Achievements

1998-2003 Clinical Medicine: Wannan Medical College
2005-2010 PhD in Pharmacology: Sun Yat-sen University
Published 16 SCI papers as first/corresponding author with a cumulative impact factor of 80

Work Experience

2010-2018 - Nanjing Medical University - Lecturer in Pharmacology
2018-present - Nanjing Medical University - Associate Professor in Pharmacology

Areas of Focus	Pathogenesis and drug targets of neurodegenerative diseases
Publications	Astragaloside IV inhibits astrocyte senescence: implication in Parkinson's disease, Xia ML, Xie XH, Ding JH, Du RH, Hu G, 2020 Kir6.1/K-ATP channel on astrocytes protects against dopaminergic neurodegeneration in the MPTP mouse model of Parkinson's disease via promoting mitophagy, Hu ZL, Sun T, Lu M, Ding JH, Du RH, Hu G, 2019 Astrocyte-specific deletion of Kir6.1/K-ATP channel aggravates cerebral ischemia/reperfusion injury through endoplasmic reticulum stress in mice, Zhong CJ, Chen MM, Lu M, Ding JH, Du RH, Hu G, 2019 The pore-forming subunit Kir6.1 of the K-ATP channel negatively regulates the NLRP3 inflammasome to control insulin resistance by interacting with NLRP3, Du RH, Lu M, Wang C, Ding JH, Wu G, Hu G, 2019 Kir6.1/K-ATP channel modulates microglia phenotypes: implication in Parkinson's disease, Du RH, Sun HB, Hu ZL, Lu M, Ding JH, Hu G, 2018 α-Synuclein disrupts the anti-inflammatory role of Drd2 via interfering β-arrestin2-TAB1 interaction in astrocytes, Du RH#, Zhou Y#, Xia ML, Lu M, Ding JH, Hu G, 2018 MicroRNA-7 targets Nod-like receptor protein 3 inflammasome to modulate neuroinflammation in the pathogenesis of Parkinson’s disease, Zhou Y#, Lu M#, Du RH#, Qiao C, Jiang CY, Zhang KZ, Ding JH, and Hu G, 2016 Uncoupling protein 2 modulation of the NLRP3 inflammasome in astrocytes and its implications in depression, Du RH#, Wu FF#, Lu M#, Shu XD, Ding JH, Wu G, Hu G, 2016 Fluoxetine Inhibits NLRP3 Inflammasome Activation: Implication in Depression, Du RH, Tan J, Sun XY, Lu M, Ding JH, Hu G, 2016 Endophilin A2 Influences Volume-Regulated Chloride Current by Mediating ClC-3 Trafficking in Vascular Smooth Muscle Cells, Liu CZ#, Li XY#, Du RH#, Gao M, Ma MM, Li FY, Huang EW, Sun HS, Wang GL, Guan YY, 2016 β-Arrestin 2 mediates the anti-inflammatory effects of fluoxetine in lipopolysaccharide-stimulated microglial cells, Du RW, Du RH, Bu WG, 2014 Kir6.2-containing ATP-sensitive K(+) channel is required for cardioprotection of resveratrol in mice, Du RH, Dai T, Cao WJ, Lu M, Ding JH, Hu G, 2014 Kir6.2 knockout aggravates lipopolysaccharide-induced mouse liver injury via enhancing NLRP3 inflammasome activation, Du RH, Tan J, Yan N, Wang L, Qiao C, Ding JH, Lu M, Hu G, 2014

Areas of Focus

Pathogenesis and drug targets of neurodegenerative diseases

Publications

Astragaloside IV inhibits astrocyte senescence: implication in Parkinson's disease, Xia ML, Xie XH, Ding JH, Du RH*, Hu G*, 2020
Kir6.1/K-ATP channel on astrocytes protects against dopaminergic neurodegeneration in the MPTP mouse model of Parkinson's disease via promoting mitophagy, Hu ZL, Sun T, Lu M, Ding JH, Du RH*, Hu G*, 2019
Astrocyte-specific deletion of Kir6.1/K-ATP channel aggravates cerebral ischemia/reperfusion injury through endoplasmic reticulum stress in mice, Zhong CJ, Chen MM, Lu M, Ding JH, Du RH*, Hu G*, 2019
The pore-forming subunit Kir6.1 of the K-ATP channel negatively regulates the NLRP3 inflammasome to control insulin resistance by interacting with NLRP3, Du RH, Lu M, Wang C, Ding JH, Wu G, Hu G*, 2019
Kir6.1/K-ATP channel modulates microglia phenotypes: implication in Parkinson's disease, Du RH, Sun HB, Hu ZL, Lu M, Ding JH, Hu G*, 2018
α-Synuclein disrupts the anti-inflammatory role of Drd2 via interfering β-arrestin2-TAB1 interaction in astrocytes, Du RH#, Zhou Y#, Xia ML, Lu M, Ding JH, Hu G, 2018
MicroRNA-7 targets Nod-like receptor protein 3 inflammasome to modulate neuroinflammation in the pathogenesis of Parkinson’s disease, Zhou Y#, Lu M#, Du RH#, Qiao C, Jiang CY, Zhang KZ, Ding JH, and Hu G*, 2016
Uncoupling protein 2 modulation of the NLRP3 inflammasome in astrocytes and its implications in depression, Du RH#, Wu FF#, Lu M#, Shu XD, Ding JH, Wu G, Hu G*, 2016
Fluoxetine Inhibits NLRP3 Inflammasome Activation: Implication in Depression, Du RH, Tan J, Sun XY, Lu M, Ding JH, Hu G, 2016
Endophilin A2 Influences Volume-Regulated Chloride Current by Mediating ClC-3 Trafficking in Vascular Smooth Muscle Cells, Liu CZ#, Li XY#, Du RH#, Gao M, Ma MM, Li FY, Huang EW, Sun HS, Wang GL, Guan YY, 2016
β-Arrestin 2 mediates the anti-inflammatory effects of fluoxetine in lipopolysaccharide-stimulated microglial cells, Du RW, Du RH*, Bu WG*, 2014
Kir6.2-containing ATP-sensitive K(+) channel is required for cardioprotection of resveratrol in mice, Du RH, Dai T, Cao WJ, Lu M, Ding JH, Hu G, 2014
Kir6.2 knockout aggravates lipopolysaccharide-induced mouse liver injury via enhancing NLRP3 inflammasome activation, Du RH, Tan J, Yan N, Wang L, Qiao C, Ding JH, Lu M, Hu G, 2014

Neurodegenerative Diseases Pathogenesis Drug Targets Neuropharmacology Parkinson'S Disease Microglia Astrocytes Inflammation Mitophagy Nlrp3 Inflammasome

Fengfei Ding - Fudan University

Ming Lu - Nanjing Medical University

Junying Gao - Nanjing Medical University

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