Professor | 杜仁红 | 数字化转型顾问

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Name	杜仁红
Contact Information	drh@njmu.edu.cn
Language ability	中文, 英语
Province	江苏

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Name

杜仁红

Contact Information

drh@njmu.edu.cn

Language ability

中文, 英语

Province

江苏

University/Lab Affiliation and Title	南京医科大学
Faculty	Basic Medical Sciences
Academic Background & Achievements	1998-2003 临床医学: 皖南医学院 2005-2010 药理学博士: 中山大学以第一作者/通讯作者发表16篇SCI论文，累计影响因子达80
Work Experience	2010-2018 - 南京医科大学 - 药理学讲师 2018-至今 - 南京医科大学 - 药理学副教授

University/Lab Affiliation and Title

南京医科大学

Faculty

Basic Medical Sciences

Academic Background & Achievements

1998-2003 临床医学: 皖南医学院
2005-2010 药理学博士: 中山大学
以第一作者/通讯作者发表16篇SCI论文，累计影响因子达80

Work Experience

2010-2018 - 南京医科大学 - 药理学讲师
2018-至今 - 南京医科大学 - 药理学副教授

Areas of Focus	神经退行性疾病的发病机制与药物靶点研究
Publications	Astragaloside IV inhibits astrocyte senescence: implication in Parkinson's disease, Xia ML, Xie XH, Ding JH, Du RH, Hu G, 2020 Kir6.1/K-ATP channel on astrocytes protects against dopaminergic neurodegeneration in the MPTP mouse model of Parkinson's disease via promoting mitophagy, Hu ZL, Sun T, Lu M, Ding JH, Du RH, Hu G, 2019 Astrocyte-specific deletion of Kir6.1/K-ATP channel aggravates cerebral ischemia/reperfusion injury through endoplasmic reticulum stress in mice, Zhong CJ, Chen MM, Lu M, Ding JH, Du RH, Hu G, 2019 The pore-forming subunit Kir6.1 of the K-ATP channel negatively regulates the NLRP3 inflammasome to control insulin resistance by interacting with NLRP3, Du RH, Lu M, Wang C, Ding JH, Wu G, Hu G, 2019 Kir6.1/K-ATP channel modulates microglia phenotypes: implication in Parkinson's disease, Du RH, Sun HB, Hu ZL, Lu M, Ding JH, Hu G, 2018 α-Synuclein disrupts the anti-inflammatory role of Drd2 via interfering β-arrestin2-TAB1 interaction in astrocytes, Du RH#, Zhou Y#, Xia ML, Lu M, Ding JH, Hu G, 2018 MicroRNA-7 targets Nod-like receptor protein 3 inflammasome to modulate neuroinflammation in the pathogenesis of Parkinson’s disease, Zhou Y#, Lu M#, Du RH#, Qiao C, Jiang CY, Zhang KZ, Ding JH, and Hu G, 2016 Uncoupling protein 2 modulation of the NLRP3 inflammasome in astrocytes and its implications in depression, Du RH#, Wu FF#, Lu M#, Shu XD, Ding JH, Wu G, Hu G, 2016 Fluoxetine Inhibits NLRP3 Inflammasome Activation: Implication in Depression, Du RH, Tan J, Sun XY, Lu M, Ding JH, Hu G, 2016 Endophilin A2 Influences Volume-Regulated Chloride Current by Mediating ClC-3 Trafficking in Vascular Smooth Muscle Cells, Liu CZ#, Li XY#, Du RH#, Gao M, Ma MM, Li FY, Huang EW, Sun HS, Wang GL, Guan YY, 2016 β-Arrestin 2 mediates the anti-inflammatory effects of fluoxetine in lipopolysaccharide-stimulated microglial cells, Du RW, Du RH, Bu WG, 2014 Kir6.2-containing ATP-sensitive K(+) channel is required for cardioprotection of resveratrol in mice, Du RH, Dai T, Cao WJ, Lu M, Ding JH, Hu G, 2014 Kir6.2 knockout aggravates lipopolysaccharide-induced mouse liver injury via enhancing NLRP3 inflammasome activation, Du RH, Tan J, Yan N, Wang L, Qiao C, Ding JH, Lu M, Hu G, 2014

Areas of Focus

神经退行性疾病的发病机制与药物靶点研究

Publications

Astragaloside IV inhibits astrocyte senescence: implication in Parkinson's disease, Xia ML, Xie XH, Ding JH, Du RH*, Hu G*, 2020
Kir6.1/K-ATP channel on astrocytes protects against dopaminergic neurodegeneration in the MPTP mouse model of Parkinson's disease via promoting mitophagy, Hu ZL, Sun T, Lu M, Ding JH, Du RH*, Hu G*, 2019
Astrocyte-specific deletion of Kir6.1/K-ATP channel aggravates cerebral ischemia/reperfusion injury through endoplasmic reticulum stress in mice, Zhong CJ, Chen MM, Lu M, Ding JH, Du RH*, Hu G*, 2019
The pore-forming subunit Kir6.1 of the K-ATP channel negatively regulates the NLRP3 inflammasome to control insulin resistance by interacting with NLRP3, Du RH, Lu M, Wang C, Ding JH, Wu G, Hu G*, 2019
Kir6.1/K-ATP channel modulates microglia phenotypes: implication in Parkinson's disease, Du RH, Sun HB, Hu ZL, Lu M, Ding JH, Hu G*, 2018
α-Synuclein disrupts the anti-inflammatory role of Drd2 via interfering β-arrestin2-TAB1 interaction in astrocytes, Du RH#, Zhou Y#, Xia ML, Lu M, Ding JH, Hu G, 2018
MicroRNA-7 targets Nod-like receptor protein 3 inflammasome to modulate neuroinflammation in the pathogenesis of Parkinson’s disease, Zhou Y#, Lu M#, Du RH#, Qiao C, Jiang CY, Zhang KZ, Ding JH, and Hu G*, 2016
Uncoupling protein 2 modulation of the NLRP3 inflammasome in astrocytes and its implications in depression, Du RH#, Wu FF#, Lu M#, Shu XD, Ding JH, Wu G, Hu G*, 2016
Fluoxetine Inhibits NLRP3 Inflammasome Activation: Implication in Depression, Du RH, Tan J, Sun XY, Lu M, Ding JH, Hu G, 2016
Endophilin A2 Influences Volume-Regulated Chloride Current by Mediating ClC-3 Trafficking in Vascular Smooth Muscle Cells, Liu CZ#, Li XY#, Du RH#, Gao M, Ma MM, Li FY, Huang EW, Sun HS, Wang GL, Guan YY, 2016
β-Arrestin 2 mediates the anti-inflammatory effects of fluoxetine in lipopolysaccharide-stimulated microglial cells, Du RW, Du RH*, Bu WG*, 2014
Kir6.2-containing ATP-sensitive K(+) channel is required for cardioprotection of resveratrol in mice, Du RH, Dai T, Cao WJ, Lu M, Ding JH, Hu G, 2014
Kir6.2 knockout aggravates lipopolysaccharide-induced mouse liver injury via enhancing NLRP3 inflammasome activation, Du RH, Tan J, Yan N, Wang L, Qiao C, Ding JH, Lu M, Hu G, 2014

神经退行性疾病发病机制药物靶点神经药理学帕金森病小胶质细胞星形胶质细胞炎症线粒体自噬 Nlrp3炎症小体

夏朋延 - 北京大学

龙志敏 - 重庆医科大学

Area of Focus
神经退行性疾病 | 发病机制 | 药物治疗 | 阿尔茨海默病 | 机制 | 研究 | 自噬 | 炎症 | Nlrp3炎症小体 | Mir-195

张培培 - 北京大学

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