Professor | Miao Yanying | Asia Growth Partners

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Name

Miao Yanying

Contact Information

yymiao@fudan.edu.cn

Language ability

Chinese, English

Province

Shanghai

University/Lab Affiliation and Title	Fudan University
Faculty	Brain Science Research Institute
Academic Background & Achievements	2007 - Medical Doctorate: Peking University Member of the Shanghai Physiological Society Committee Member of the Chinese Research Hospital Association's Neuroregeneration and Repair Committee
Work Experience	2007-present - Lecturer, Fudan University Institute of Neurobiology 2009-present - Associate Researcher, Fudan University Institute of Brain Science 2012-2013 - Visiting Scholar, University of Chicago

University/Lab Affiliation and Title

Fudan University

Faculty

Brain Science Research Institute

Academic Background & Achievements

2007 - Medical Doctorate: Peking University
Member of the Shanghai Physiological Society
Committee Member of the Chinese Research Hospital Association's Neuroregeneration and Repair Committee

Work Experience

2007-present - Lecturer, Fudan University Institute of Neurobiology
2009-present - Associate Researcher, Fudan University Institute of Brain Science
2012-2013 - Visiting Scholar, University of Chicago

Areas of Focus	Retinal Physiology and Related Diseases Mechanisms of Retinal Ganglion Cell Damage in Glaucoma
Publications	Modulation of Rac1/PAK1/connexin43-mediated ATP release from astrocytes contributes to retinal ganglion cell survival in experimental glaucoma., Zhao GL, Zhou H, Guo YH, Zhong SM, Zhou H, Li F, Lei B, Wang Z, Miao Y, 2023 Activation of retinal glial cells contributes to the degeneration of ganglion cells in experimental glaucoma., Miao Y#, Zhao GL#, Cheng S, Wang Z, Yang XL, 2023 Rac1 conditional deletion attenuates retinal ganglion cell apoptosis by accelerating autophagic flux in a mouse model of chronic ocular hypertension., Zhang ML#, Zhao GL#, Hou Y, Zhong SM, Xu LJ, Li F, Niu WR, Yuan F, Yang XL, Wang Z, Miao Y, 2020 EphrinB/EphB forward signaling in Muller cells causes apoptosis of retinal ganglion cells by increasing tumor necrosis factor alpha production in rat experimental glaucomatous model., Liu ST, Zhong SM, Li XY, Gao F, Li F, Zhang ML, Zhu K, Sun XH, Wang X, Miao Y, Yang XL, Wang Z, 2018 Dopamine D1 receptor-mediated upregulation of BKCa currents modifies Müller cell gliosis in a rat chronic ocular hypertension model., Wu HJ#, Li XY#, Qian WJ, Li Q, Wang SY, Ji M, Ma YY, Gao F, Sun XH, Wang X, Miao Y, Yang XL, Wang Z, 2018

Areas of Focus

Retinal Physiology and Related Diseases
Mechanisms of Retinal Ganglion Cell Damage in Glaucoma

Publications

Modulation of Rac1/PAK1/connexin43-mediated ATP release from astrocytes contributes to retinal ganglion cell survival in experimental glaucoma., Zhao GL, Zhou H, Guo YH, Zhong SM, Zhou H, Li F, Lei B, Wang Z*, Miao Y*, 2023
Activation of retinal glial cells contributes to the degeneration of ganglion cells in experimental glaucoma., Miao Y#, Zhao GL#, Cheng S, Wang Z*, Yang XL*, 2023
Rac1 conditional deletion attenuates retinal ganglion cell apoptosis by accelerating autophagic flux in a mouse model of chronic ocular hypertension., Zhang ML#, Zhao GL#, Hou Y, Zhong SM, Xu LJ, Li F, Niu WR, Yuan F, Yang XL, Wang Z*, Miao Y*, 2020
EphrinB/EphB forward signaling in Muller cells causes apoptosis of retinal ganglion cells by increasing tumor necrosis factor alpha production in rat experimental glaucomatous model., Liu ST, Zhong SM, Li XY, Gao F, Li F, Zhang ML, Zhu K, Sun XH, Wang X, Miao Y*, Yang XL, Wang Z*, 2018
Dopamine D1 receptor-mediated upregulation of BKCa currents modifies Müller cell gliosis in a rat chronic ocular hypertension model., Wu HJ#, Li XY#, Qian WJ, Li Q, Wang SY, Ji M, Ma YY, Gao F, Sun XH, Wang X, Miao Y*, Yang XL, Wang Z*, 2018

Retina Disease Glaucoma Neuroprotection Autophagy Apoptosis Glial Cells Microenvironment Small Molecules Research

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Shijun Weng - Fudan University

Xu Gezhi - Fudan University

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