Professor | Kai-Rui Mao | Asia Growth Partners

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Name	Kai-Rui Mao
Contact Information	maok@xmu.edu.cn
Language ability	Chinese, English
Province	Fujian

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Name

Kai-Rui Mao

Contact Information

maok@xmu.edu.cn

Language ability

Chinese, English

Province

Fujian

University/Lab Affiliation and Title	Xiamen University
Faculty	Medical
Academic Background & Achievements	2011 - PhD in Science: Shanghai Institute of Biochemistry and Cell Biology, Chinese Academy of Sciences 2004 - Bachelor of Science in Biotechnology: Shandong Normal University
Work Experience	2019-present - Xiamen University, School of Life Sciences, Minjiang Scholar Distinguished Professor, Head of Mucosal Immunology Group 2013-2019 - Visiting Scholar at National Institute of Allergy and Infectious Diseases (NIAID), NIH, USA 2011-2013 - Research Assistant at Shanghai Institute of Biochemistry and Cell Biology, Chinese Academy of Sciences

University/Lab Affiliation and Title

Xiamen University

Faculty

Medical

Academic Background & Achievements

2011 - PhD in Science: Shanghai Institute of Biochemistry and Cell Biology, Chinese Academy of Sciences
2004 - Bachelor of Science in Biotechnology: Shandong Normal University

Work Experience

2019-present - Xiamen University, School of Life Sciences, Minjiang Scholar Distinguished Professor, Head of Mucosal Immunology Group
2013-2019 - Visiting Scholar at National Institute of Allergy and Infectious Diseases (NIAID), NIH, USA
2011-2013 - Research Assistant at Shanghai Institute of Biochemistry and Cell Biology, Chinese Academy of Sciences

Areas of Focus	Interaction between gut microbiota and immune system
Publications	Thinking differently about ILCs-Not just tissue resident and not just the same as CD4(+) T-cell effectors, Huang, Y., Mao, K., and Germain, R. N., 2018 Innate and adaptive lymphocytes sequentially shape the gut microbiota and lipid metabolism, Mao, K., Baptista, A. P., Tamoutounour, S., Zhuang, L., Bouladoux, N., Martins, A. J., Huang, Y., Gerner, M. Y., Belkaid, Y., and Germain, R. N., 2018 S1P-dependent interorgan trafficking of group 2 innate lymphoid cells supports host defense, Huang, Y., Mao, K., Chen, X., Sun, M. A., Kawabe, T., Li, W., Usher, N., Zhu, J., Urban, J. F., Jr., Paul, W. E., and Germain, R. N., 2018 Nitric oxide suppresses NLRP3 inflammasome activation and protects against LPS-induced septic shock, Mao, K., Chen, S., Chen, M., Ma, Y., Wang, Y., Huang, B., He, Z., Zeng, Y., Hu, Y., Sun, S., Li, J., Wu, X., Wang, X., Strober, W., Chen, C., Meng, G., and Sun, B., 2013 beta-arrestin1 is critical for the full activation of NLRP3 and NLRC4 inflammasomes, Mao, K., Chen, S., Wang, Y., Zeng, Y., Ma, Y., Hu, Y., Zhang, H., Sun, S., Wu, X., Meng, G., Pei, G., and Sun, B., 2015 Group 3 innate lymphoid cells continuously require the transcription factor GATA-3 after commitment, Zhong, C., Cui, K., Wilhelm, C., Hu, G., Mao, K., Belkaid, Y., Zhao, K., and Zhu, J., 2016 Cellular localization of NLRP3 inflammasome, Wang, Y., Yang, C., Mao, K., Chen, S., Meng, G., and Sun, B., 2013 Tripartite-motif protein 30 negatively regulates NLRP3 inflammasome activation by modulating reactive oxygen species production, Hu, Y., Mao, K., Zeng, Y., Chen, S., Tao, Z., Yang, C., Sun, S., Wu, X., Meng, G., and Sun, B., 2010 Trichosanthin functions as Th2-type adjuvant in induction of allergic airway inflammation, Wang, Y., Mao, K., Sun, S., Lin, G., Wu, X., Yao, G., and Sun, B., 2009 TRIM30 alpha negatively regulates TLR-mediated NF-kappa B activation by targeting TAB2 and TAB3 for degradation, Shi, M., Deng, W., Bi, E., Mao, K., Ji, Y., Lin, G., Wu, X., Tao, Z., Li, Z., Cai, X., Sun, S., Xiang, C., and Sun, B., 2008

Areas of Focus

Interaction between gut microbiota and immune system

Publications

Thinking differently about ILCs-Not just tissue resident and not just the same as CD4(+) T-cell effectors, Huang, Y., Mao, K., and Germain, R. N., 2018
Innate and adaptive lymphocytes sequentially shape the gut microbiota and lipid metabolism, Mao, K., Baptista, A. P., Tamoutounour, S., Zhuang, L., Bouladoux, N., Martins, A. J., Huang, Y., Gerner, M. Y., Belkaid, Y., and Germain, R. N., 2018
S1P-dependent interorgan trafficking of group 2 innate lymphoid cells supports host defense, Huang, Y., Mao, K., Chen, X., Sun, M. A., Kawabe, T., Li, W., Usher, N., Zhu, J., Urban, J. F., Jr., Paul, W. E., and Germain, R. N., 2018
Nitric oxide suppresses NLRP3 inflammasome activation and protects against LPS-induced septic shock, Mao, K., Chen, S., Chen, M., Ma, Y., Wang, Y., Huang, B., He, Z., Zeng, Y., Hu, Y., Sun, S., Li, J., Wu, X., Wang, X., Strober, W., Chen, C., Meng, G., and Sun, B., 2013
beta-arrestin1 is critical for the full activation of NLRP3 and NLRC4 inflammasomes, Mao, K., Chen, S., Wang, Y., Zeng, Y., Ma, Y., Hu, Y., Zhang, H., Sun, S., Wu, X., Meng, G., Pei, G., and Sun, B., 2015
Group 3 innate lymphoid cells continuously require the transcription factor GATA-3 after commitment, Zhong, C., Cui, K., Wilhelm, C., Hu, G., Mao, K., Belkaid, Y., Zhao, K., and Zhu, J., 2016
Cellular localization of NLRP3 inflammasome, Wang, Y., Yang, C., Mao, K., Chen, S., Meng, G., and Sun, B., 2013
Tripartite-motif protein 30 negatively regulates NLRP3 inflammasome activation by modulating reactive oxygen species production, Hu, Y., Mao, K., Zeng, Y., Chen, S., Tao, Z., Yang, C., Sun, S., Wu, X., Meng, G., and Sun, B., 2010
Trichosanthin functions as Th2-type adjuvant in induction of allergic airway inflammation, Wang, Y., Mao, K., Sun, S., Lin, G., Wu, X., Yao, G., and Sun, B., 2009
TRIM30 alpha negatively regulates TLR-mediated NF-kappa B activation by targeting TAB2 and TAB3 for degradation, Shi, M., Deng, W., Bi, E., Mao, K., Ji, Y., Lin, G., Wu, X., Tao, Z., Li, Z., Cai, X., Sun, S., Xiang, C., and Sun, B., 2008

Gut Microbiota Immune System Physiological Response Disease Development Disease Treatment Imaging Systems Immunology Homeostasis Epithelial Stem Cell Differentiation Immune Inflammation

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