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毛开睿
maok@xmu.edu.cn
中文, 英文
福建
厦门大学
Medical
  • 2011年 - 理学博士: 中国科学院上海生命科学研究院生化细胞所
  • 2004年 - 生物技术理学学士: 山东师范大学
  • 2019年至今 - 厦门大学生命科学学院,闽江学者特聘教授,黏膜免疫课题组组长
  • 2013-2019年 - 美国国立卫生研究院(NIH)过敏和感染性疾病研究所(NIAID),访问学者
  • 2011-2013年 - 中国科学院上海生命科学研究院生化细胞所,研究助理
肠道菌群和免疫系统之间的相互作用
  • Thinking differently about ILCs-Not just tissue resident and not just the same as CD4(+) T-cell effectors, Huang, Y., Mao, K., and Germain, R. N., 2018
  • Innate and adaptive lymphocytes sequentially shape the gut microbiota and lipid metabolism, Mao, K., Baptista, A. P., Tamoutounour, S., Zhuang, L., Bouladoux, N., Martins, A. J., Huang, Y., Gerner, M. Y., Belkaid, Y., and Germain, R. N., 2018
  • S1P-dependent interorgan trafficking of group 2 innate lymphoid cells supports host defense, Huang, Y., Mao, K., Chen, X., Sun, M. A., Kawabe, T., Li, W., Usher, N., Zhu, J., Urban, J. F., Jr., Paul, W. E., and Germain, R. N., 2018
  • Nitric oxide suppresses NLRP3 inflammasome activation and protects against LPS-induced septic shock, Mao, K., Chen, S., Chen, M., Ma, Y., Wang, Y., Huang, B., He, Z., Zeng, Y., Hu, Y., Sun, S., Li, J., Wu, X., Wang, X., Strober, W., Chen, C., Meng, G., and Sun, B., 2013
  • beta-arrestin1 is critical for the full activation of NLRP3 and NLRC4 inflammasomes, Mao, K., Chen, S., Wang, Y., Zeng, Y., Ma, Y., Hu, Y., Zhang, H., Sun, S., Wu, X., Meng, G., Pei, G., and Sun, B., 2015
  • Group 3 innate lymphoid cells continuously require the transcription factor GATA-3 after commitment, Zhong, C., Cui, K., Wilhelm, C., Hu, G., Mao, K., Belkaid, Y., Zhao, K., and Zhu, J., 2016
  • Cellular localization of NLRP3 inflammasome, Wang, Y., Yang, C., Mao, K., Chen, S., Meng, G., and Sun, B., 2013
  • Tripartite-motif protein 30 negatively regulates NLRP3 inflammasome activation by modulating reactive oxygen species production, Hu, Y., Mao, K., Zeng, Y., Chen, S., Tao, Z., Yang, C., Sun, S., Wu, X., Meng, G., and Sun, B., 2010
  • Trichosanthin functions as Th2-type adjuvant in induction of allergic airway inflammation, Wang, Y., Mao, K., Sun, S., Lin, G., Wu, X., Yao, G., and Sun, B., 2009
  • TRIM30 alpha negatively regulates TLR-mediated NF-kappa B activation by targeting TAB2 and TAB3 for degradation, Shi, M., Deng, W., Bi, E., Mao, K., Ji, Y., Lin, G., Wu, X., Tao, Z., Li, Z., Cai, X., Sun, S., Xiang, C., and Sun, B., 2008
肠道菌群 免疫系统 生理反应 疾病发展 疾病治疗 成像系统 免疫学 稳态 上皮干细胞分化 免疫炎症

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