Professor | Li Baojie | Asia Growth Partners

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Name	Li Baojie
Contact Information	libj.bio@gmail.com
Language ability	Chinese, English
Province	Shanghai

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Name

Li Baojie

Contact Information

libj.bio@gmail.com

Language ability

Chinese, English

Province

Shanghai

University/Lab Affiliation and Title	Shanghai Jiao Tong University
Faculty	Life Sciences
Academic Background & Achievements	Bachelor's Degree: Beijing Normal University PhD: Albert Einstein College of Medicine, USA Postdoctoral Research: Columbia University Recipient of Shanghai 'Pujiang Talent Plan' 2009 Key Project Leader in 863 Program 'Comprehensive Platform Technology of Whole Genome Association Analysis'
Work Experience	2001-2009 - Institute of Molecular and Cell Biology, Singapore - PI, Senior PI

University/Lab Affiliation and Title

Shanghai Jiao Tong University

Faculty

Life Sciences

Academic Background & Achievements

Bachelor's Degree: Beijing Normal University
PhD: Albert Einstein College of Medicine, USA
Postdoctoral Research: Columbia University
Recipient of Shanghai 'Pujiang Talent Plan' 2009
Key Project Leader in 863 Program 'Comprehensive Platform Technology of Whole Genome Association Analysis'

Work Experience

2001-2009 - Institute of Molecular and Cell Biology, Singapore - PI, Senior PI

Areas of Focus	Mesenchymal stem cell in vivo self-renewal, differentiation, and aging regulation mechanisms Bone development, bone metabolism, and pathogenesis of osteoporosis DNA damage stress response, aging, and tumor occurrence
Publications	A positive role for c-Abl in Atm and Atr activation in DNA damage response, Wang X, Zeng L, Wang J, Chau JF, Lai KP, Jia D, Poonepalli A, Hande MP, Liu H, He G, He L, Li B, 2011 S6K1 is a multifaceted regulator of Mdm2 that connects nutrient status and DNA damage response, Lai KP, Leong WF, Chau JF, Jia D, Zeng L, Liu H, He L, Hao A, Zhang H, Meek D, Velagapudi C, Habib SL, Li B, 2010 Atm deficient mice: an osteoporosis model with defective osteoblast differentiation and increased osteoblastogenesis, Rasheed N, Wang X, Niu QT, Yeh J, Li B, 2006 p53 represses the expression of transcription factor osterix and functions as a negative regulator of osteoblastogenesis and bone formation, Wang X, Kua HY, Hu Y, Guo K, Zeng Q, Wu Q, Ng HH, Karsenty G, de Crombrugghe B, Yeh J, Li B, 2006 Distinct roles for c-Abl and Atm in Prx I induction by modulating expression of protein kinase C, Li B, Wang X, Rasheed N, Hu Y, Boast S, Ishii T, Nakayama K, Nakayama KI, Goff SP, 2004 Activation of p38MAPK is required for osteoblast differentiation, Hu Y, Chan E, Wang X, Li B, 2003 c-Abl knockout mice are osteoporotic and show defects in osteoblast maturation, Li B, Boast B, de los Santos K, Schieren I, Quiroz M, Teitelbaum SL, Tondravi MM, Goff SP, 2000

Areas of Focus

Mesenchymal stem cell in vivo self-renewal, differentiation, and aging regulation mechanisms
Bone development, bone metabolism, and pathogenesis of osteoporosis
DNA damage stress response, aging, and tumor occurrence

Publications

A positive role for c-Abl in Atm and Atr activation in DNA damage response, Wang X, Zeng L, Wang J, Chau JF, Lai KP, Jia D, Poonepalli A, Hande MP, Liu H, He G, He L, Li B, 2011
S6K1 is a multifaceted regulator of Mdm2 that connects nutrient status and DNA damage response, Lai KP, Leong WF, Chau JF, Jia D, Zeng L, Liu H, He L, Hao A, Zhang H, Meek D, Velagapudi C, Habib SL, Li B, 2010
Atm deficient mice: an osteoporosis model with defective osteoblast differentiation and increased osteoblastogenesis, Rasheed N, Wang X, Niu QT, Yeh J, Li B, 2006
p53 represses the expression of transcription factor osterix and functions as a negative regulator of osteoblastogenesis and bone formation, Wang X, Kua HY, Hu Y, Guo K, Zeng Q, Wu Q, Ng HH, Karsenty G, de Crombrugghe B, Yeh J, Li B, 2006
Distinct roles for c-Abl and Atm in Prx I induction by modulating expression of protein kinase C, Li B, Wang X, Rasheed N, Hu Y, Boast S, Ishii T, Nakayama K, Nakayama KI, Goff SP, 2004
Activation of p38MAPK is required for osteoblast differentiation, Hu Y, Chan E, Wang X, Li B, 2003
c-Abl knockout mice are osteoporotic and show defects in osteoblast maturation, Li B, Boast B, de los Santos K, Schieren I, Quiroz M, Teitelbaum SL, Tondravi MM, Goff SP, 2000

Stem Cells Self-Renewal Differentiation Aging Bone Development Bone Metabolism Osteoporosis Dna Damage Stress Response Tumor

Han Song Deng - Tongji University

Zhiyong Mao - Tongji University

Fanglin Sun - Tongji University

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