Professor | Liu Yi | Asia Growth Partners

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Name	Liu Yi
Contact Information	liuyee@fudan.edu.cn
Language ability	Chinese, English
Province	Shanghai

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Name

Liu Yi

Contact Information

liuyee@fudan.edu.cn

Language ability

Chinese, English

Province

Shanghai

University/Lab Affiliation and Title	Fudan University
Faculty	Basic Medical Sciences
Academic Background & Achievements	1992.09—1997.07 Bachelor's in Genetics: Fudan University 1998.09—2004.03 Ph.D. in Biomedical Sciences: University of California, Riverside
Work Experience	2004.04—2005.08 - University of California, Riverside - Postdoctoral Researcher 2005.09—2010.01 - The Salk Institute - Postdoctoral Researcher 2010.02—2017.08 - Institute for Nutritional Sciences, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences - Researcher 2017.09—Present - School of Basic Medical Sciences, Fudan University - Researcher
Awards	2013: National Natural Science Foundation of China Excellent Young Scientists Fund 2010: Shanghai Pujiang Talent Program

University/Lab Affiliation and Title

Fudan University

Faculty

Basic Medical Sciences

Academic Background & Achievements

1992.09—1997.07 Bachelor's in Genetics: Fudan University
1998.09—2004.03 Ph.D. in Biomedical Sciences: University of California, Riverside

Work Experience

2004.04—2005.08 - University of California, Riverside - Postdoctoral Researcher
2005.09—2010.01 - The Salk Institute - Postdoctoral Researcher
2010.02—2017.08 - Institute for Nutritional Sciences, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences - Researcher
2017.09—Present - School of Basic Medical Sciences, Fudan University - Researcher

Awards

2013: National Natural Science Foundation of China Excellent Young Scientists Fund
2010: Shanghai Pujiang Talent Program

Areas of Focus	Molecular mechanisms of the interaction between circadian rhythms and glucose and lipid metabolism
Publications	A propolis-derived small molecule ameliorates metabolic syndrome in obese mice by targeting the CREB/CRTC2 transcriptional complex., Chen Y, Wang J, Wang Y, Wang P, Zhou Z, Wu R, Xu Q, You H, Liu Y, Wang L, Zhou L, Wu Y, Hu L, Liu H, Liu Y., 2022 The Circadian Protein Period2 Suppresses mTORC1 Activity via Recruiting Tsc1 to mTORC1 Complex., Wu R., Dang F., Li P., Wang P., Xu Q., Liu Z., Li Y., Wu Y., Chen Y., Liu Y., 2019 Fasting and Feeding Signals Control the Oscillatory Expression of Angptl8 to Modulate Lipid Metabolism., Dang F., Wu R., Wang P., Wu Y., Azam S., Liu Y., 2016 Insulin post-transcriptionally modulates Bmal1 protein to affect the hepatic circadian clock., Dang, F., Sun, X., Ma, X., Wu, R., Zhang, D., Chen, Y., Xu, Q., Wu, Y., Liu Y., 2016 RBP4 functions as a hepatokine in the regulation of glucose metabolism by the circadian clock in mice., Ma X, Zhou Z, Chen Y, Wu Y, Liu Y., 2016 Glucagon-CREB/CRTC2 Signaling Cascade Regulates Hepatic BMAL1 Protein., Sun, X., Dang, F., Zhang, D., Yuan, Y., Zhang, C., Wu, Y., Wang, Y., Liu, Y., 2015 Cryptochrome mediates circadian regulation of cAMP signaling and hepatic gluconeogenesis., Zhang, E.E., Liu, Y., Dentin, R., Pongsawakul, P.Y., Liu, A.C., Hirota, T., Nusinow, D.A., Sun, X., Landais, S., Kodama, Y., Brenner, D.A., Montminy, M., Kay, S.A., 2010 Targeted disruption of the CREB coactivator Crtc2 increases insulin sensitivity., Wang, Y., Inoue, H., Ravnskjaer, K., Viste, K., Miller, N., Liu, Y., Hedrick, S., Vera, L., Montminy, M., 2010 A fasting inducible switch modulates gluconeogenesis via activator/coactivator exchange., Liu, Y., Dentin, R., Chen, D., Hedrick, S., Ravnskjaer, K., Schenk, S., Milne, J., Meyers, D.J., Cole, P., Yates, J.R. 3rd., Olefsky, J., Guarente, L., Montminy, M., 2008 Insulin Modulates Gluconeogenesis via Inhibition of the Coactivator TORC2., Dentin, R., Liu, Y*., Koo, S., Hedrick, S., Vargas, T., Heredia, J., Yates, J.R. 3rd., Montminy, M., 2007

Areas of Focus

Molecular mechanisms of the interaction between circadian rhythms and glucose and lipid metabolism

Publications

A propolis-derived small molecule ameliorates metabolic syndrome in obese mice by targeting the CREB/CRTC2 transcriptional complex., Chen Y, Wang J, Wang Y, Wang P, Zhou Z, Wu R, Xu Q, You H, Liu Y, Wang L, Zhou L, Wu Y, Hu L, Liu H, Liu Y*., 2022
The Circadian Protein Period2 Suppresses mTORC1 Activity via Recruiting Tsc1 to mTORC1 Complex., Wu R., Dang F., Li P., Wang P., Xu Q., Liu Z., Li Y., Wu Y., Chen Y., Liu Y*., 2019
Fasting and Feeding Signals Control the Oscillatory Expression of Angptl8 to Modulate Lipid Metabolism., Dang F., Wu R., Wang P., Wu Y., Azam S., Liu Y*., 2016
Insulin post-transcriptionally modulates Bmal1 protein to affect the hepatic circadian clock., Dang, F., Sun, X., Ma, X., Wu, R., Zhang, D., Chen, Y., Xu, Q., Wu, Y., Liu Y*., 2016
RBP4 functions as a hepatokine in the regulation of glucose metabolism by the circadian clock in mice., Ma X, Zhou Z, Chen Y, Wu Y, Liu Y*., 2016
Glucagon-CREB/CRTC2 Signaling Cascade Regulates Hepatic BMAL1 Protein., Sun, X., Dang, F., Zhang, D., Yuan, Y., Zhang, C., Wu, Y., Wang, Y., Liu, Y*., 2015
Cryptochrome mediates circadian regulation of cAMP signaling and hepatic gluconeogenesis., Zhang, E.E*., Liu, Y*., Dentin, R., Pongsawakul, P.Y., Liu, A.C., Hirota, T., Nusinow, D.A., Sun, X., Landais, S., Kodama, Y., Brenner, D.A., Montminy, M., Kay, S.A., 2010
Targeted disruption of the CREB coactivator Crtc2 increases insulin sensitivity., Wang, Y., Inoue, H., Ravnskjaer, K., Viste, K., Miller, N., Liu, Y*., Hedrick, S., Vera, L., Montminy, M., 2010
A fasting inducible switch modulates gluconeogenesis via activator/coactivator exchange., Liu, Y*., Dentin, R.*, Chen, D., Hedrick, S., Ravnskjaer, K., Schenk, S., Milne, J., Meyers, D.J., Cole, P., Yates, J.R. 3rd., Olefsky, J., Guarente, L., Montminy, M., 2008
Insulin Modulates Gluconeogenesis via Inhibition of the Coactivator TORC2., Dentin, R.*, Liu, Y*., Koo, S., Hedrick, S., Vargas, T., Heredia, J., Yates, J.R. 3rd., Montminy, M., 2007

Circadian Rhythms Glucose Metabolism Lipid Metabolism Molecular Mechanisms Diabetes Pathophysiology Regulatory Networks Homeostasis Targeted Compounds Blood Sugar

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