Professor | Yingying Xu | Asia Growth Partners

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Name	Yingying Xu
Contact Information	nyingyingxu@fudan.edu.cn
Language ability	Chinese, English
Province	Shanghai

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Name

Yingying Xu

Contact Information

nyingyingxu@fudan.edu.cn

Language ability

Chinese, English

Province

Shanghai

University/Lab Affiliation and Title	Fudan University
Faculty	Basic Medical Sciences
Academic Background & Achievements	1996.09-2000.07 Bachelor's Degree in Pharmaceutical Chemistry: Fudan University 2001.09-2005.07 Master's Degree in Biochemistry and Molecular Biology: Fudan University 2006.09-2010.07 Ph.D. in Biochemistry and Molecular Biology: Fudan University
Work Experience	2000-2005 - Fudan University, Shanghai Medical College, Department of Biochemistry and Molecular Biology - Teaching Assistant 2006-2015 - Fudan University, Shanghai Medical College, Department of Biochemistry and Molecular Biology - Lecturer 2016-present - Fudan University, Shanghai Medical College, Department of Biochemistry and Molecular Biology - Associate Professor
Awards	Shanghai Science and Technology Progress Award (2007): Third Prize for the regulation of cell biological behavior by glycosyltransferase

University/Lab Affiliation and Title

Fudan University

Faculty

Basic Medical Sciences

Academic Background & Achievements

1996.09-2000.07 Bachelor's Degree in Pharmaceutical Chemistry: Fudan University
2001.09-2005.07 Master's Degree in Biochemistry and Molecular Biology: Fudan University
2006.09-2010.07 Ph.D. in Biochemistry and Molecular Biology: Fudan University

Work Experience

2000-2005 - Fudan University, Shanghai Medical College, Department of Biochemistry and Molecular Biology - Teaching Assistant
2006-2015 - Fudan University, Shanghai Medical College, Department of Biochemistry and Molecular Biology - Lecturer
2016-present - Fudan University, Shanghai Medical College, Department of Biochemistry and Molecular Biology - Associate Professor

Awards

Shanghai Science and Technology Progress Award (2007): Third Prize for the regulation of cell biological behavior by glycosyltransferase

Areas of Focus	Cell Polarity and Cancer Cancer Metabolism
Publications	A plasma SNORD33 signature predicts platinum benefit in metastatic triple-negative breast cancer patients, Biyun Wang, Yannan Zhao, Yi Li, Yingying Xu, Yun Chen, Qiuyu Jiang, Dingjin Yao, Li Zhang, Xichun Hu, Chaowei Fu, Si Zhang, She Chen, 2022 Loss of polarity protein Par3, via transcription factor Snail, promotes bladder cancer metastasis, Wang S, Cai J, Zhang S, Dong M, Zhang L, Xu Y, Shen B, Chen S, 2021 Polarity protein Par3 sensitizes breast cancer to paclitaxel by promoting cell cycle arrest, Yannan Zhao, Huitong Peng, Limiao Liang, Yi Li, Xichun Hu, Biyun Wang, Yingying Xu, She Chen, 2022 CAPS1 promotes colorectal cancer metastasis via Snail mediated epithelial mesenchymal transformation, Zhao Guang-Xi, Xu Ying-Ying, Weng Shu-Qiang, Zhang Si, Chen Ying, Shen Xi-Zhong, Dong Ling, Chen She, 2019 Acetylation targets HSD17B4 for degradation via the CMA pathway in response to estrone, Zhang Y, Xu YY, Yao CB, Li JT, Zhao XN, Yang HB, Zhang M, Yin M, Chen J, Lei QY, 2017 Acetylation of MAT IIa represses tumour cell growth and is decreased in human hepatocellular cancer, Yang HB, Xu YY, Zhao XN, Zou SW, Zhang Y, Zhang M, Li JT, Ren F, Wang LY, Lei QY, 2015 Cullin 3 targets methionine adenosyltransferase IIα for ubiquitylation-mediated degradation and regulates colorectal cancer cell proliferation, Wang J, Zhu ZH, Yang HB, Zhang Y, Zhao XN, Zhang M, Liu YB, Xu YY, Lei QY, 2016 All-trans-retinoic acid intensifies endoplasmic reticulum stress in N-acetylglucosaminyltransferase V repressed human hepatocarcinoma cells by perturbing homocysteine metabolism, Xu YY, Guan DY, Yang M, Wang H, Shen ZH, 2010 Apoptosis induced by all-trans retinoic acid in N-acetylglucosaminyltransferase V repressed human hepatocarcinoma cells is mediated through endoplasmic reticulum stress, Xu YY, Lu Y, Fan KY, Shen ZH, 2007 Blocking of N-acetylglucosaminyltransferase V induces cellular endoplasmic reticulum stress in human hepatocarcinoma 7721 cells, Fang H, Huang W, Xu YY, Shen ZH, Wu CQ, Qiao SY, Xu Y, Yu L, Chen HL, 2006

Areas of Focus

Cell Polarity and Cancer
Cancer Metabolism

Publications

A plasma SNORD33 signature predicts platinum benefit in metastatic triple-negative breast cancer patients, Biyun Wang, Yannan Zhao, Yi Li, Yingying Xu, Yun Chen, Qiuyu Jiang, Dingjin Yao, Li Zhang, Xichun Hu, Chaowei Fu, Si Zhang, She Chen, 2022
Loss of polarity protein Par3, via transcription factor Snail, promotes bladder cancer metastasis, Wang S, Cai J, Zhang S, Dong M, Zhang L, Xu Y, Shen B, Chen S, 2021
Polarity protein Par3 sensitizes breast cancer to paclitaxel by promoting cell cycle arrest, Yannan Zhao, Huitong Peng, Limiao Liang, Yi Li, Xichun Hu, Biyun Wang, Yingying Xu, She Chen, 2022
CAPS1 promotes colorectal cancer metastasis via Snail mediated epithelial mesenchymal transformation, Zhao Guang-Xi, Xu Ying-Ying, Weng Shu-Qiang, Zhang Si, Chen Ying, Shen Xi-Zhong, Dong Ling, Chen She, 2019
Acetylation targets HSD17B4 for degradation via the CMA pathway in response to estrone, Zhang Y, Xu YY, Yao CB, Li JT, Zhao XN, Yang HB, Zhang M, Yin M, Chen J, Lei QY, 2017
Acetylation of MAT IIa represses tumour cell growth and is decreased in human hepatocellular cancer, Yang HB, Xu YY, Zhao XN, Zou SW, Zhang Y, Zhang M, Li JT, Ren F, Wang LY, Lei QY, 2015
Cullin 3 targets methionine adenosyltransferase IIα for ubiquitylation-mediated degradation and regulates colorectal cancer cell proliferation, Wang J, Zhu ZH, Yang HB, Zhang Y, Zhao XN, Zhang M, Liu YB, Xu YY, Lei QY, 2016
All-trans-retinoic acid intensifies endoplasmic reticulum stress in N-acetylglucosaminyltransferase V repressed human hepatocarcinoma cells by perturbing homocysteine metabolism, Xu YY, Guan DY, Yang M, Wang H, Shen ZH, 2010
Apoptosis induced by all-trans retinoic acid in N-acetylglucosaminyltransferase V repressed human hepatocarcinoma cells is mediated through endoplasmic reticulum stress, Xu YY, Lu Y, Fan KY, Shen ZH, 2007
Blocking of N-acetylglucosaminyltransferase V induces cellular endoplasmic reticulum stress in human hepatocarcinoma 7721 cells, Fang H, Huang W, Xu YY, Shen ZH, Wu CQ, Qiao SY, Xu Y, Yu L, Chen HL, 2006

Cell Polarity Cancer Tumor Metabolism Biochemistry Molecular Biology Glycosyltransferase Cell Behavior Apoptosis Breast Cancer

Tanjun Tong - Peking University

Yingyu Chen - Peking University

Chunlan Yan - Zhejiang University

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