蒋德伟
jiangdewei@mail.kiz.ac.cn
中文, 英语
云南
中国科学院大学
Medical School
  • 2013-09--2016-07 理学博士: 中国科学院大学
  • 2010-09--2013-07 理学硕士: 云南大学
  • 2006-09--2010-07 理学学士: 云南大学
  • 2022-01~现在 - 中国科学院昆明动物研究所 - 研究员
  • 2017-09~2021-12 - 中国科学院昆明动物研究所 - 副研究员
  • 2016-07~2017-09 - 中国科学院昆明动物研究所 - 助理研究员
表观遗传调控与肿瘤
代谢、DNA损伤应答与肿瘤
肿瘤生物治疗
  • DNA damage chemotherapeutic drugs suppress basal-like breast cancer growth by down-regulating the transcription of the FOXO1-KLF5 axis., Cui, Qiuxia, Sun, Jian, Yuan, Jingping, Li, Juanjuan, Yang, Chuanyu, Du, Guangshi, Zhou, Chengang, Qiu, Pu, Gao, Jidong, Zhang, Yuanqi, Jiang, Dewei, Chen, Ceshi., 2024
  • Targeting the KLF5-EphA2 axis can restrain cancer stemness and overcome chemoresistance in basal-like breast cancer., Zhao, Ping, Sun, Jian, Huang, Xinwei, Zhang, Xiangwu, Liu, Xin, Liu, Rong, Du, Guangshi, Gan, Wenqiang, Yang, Chuanyu, Tang, Yiyin, Chen, Ceshi, Jiang, Dewei., 2023
  • The tumor-nerve circuit in breast cancer., Cui, Qiuxia, Jiang, Dewei, Zhang, Yuanqi, Chen, Ceshi., 2023
  • YB-1 is a positive regulator of KLF5 transcription factor in basal-like breast cancer., Dewei Jiang, Ting Qiu, Junjiang Peng, Siyuan Li, Tala, Wenlong Ren, Chuanyu Yang, Yi Wen, Chuan-Huizi Chen, Jian Sun, Yingying Wu, Rong Liu, Jun Zhou, Kongming Wu, Wen Liu, Xioayun Mao, Zhongmei Zhou, Ceshi Chen., 2022
  • A feedforward circuit between KLF5 and lncRNA KPRT4 contributes to BLBCs., Guanghsi Du, Jian Sun, Zhen Li, Qian Zhang, Wenjing Liu, Chuanyu Yang, Ping Zhao, Xingye Wang, Qiyan Yin, Yao Luo, Jinhuan Song, Yi Wen, Haioxia Wang, Chuan-Huizi Chen, Guosheng Hu, Zhongmei Zhou, Xiaoyun Mao, Wen Liu, Zhenzhen Liu, Dewei Jiang, Ceshi Chen., 2022
  • RNF126-Mediated MRE11 Ubiquitination Activates the DNA Damage Response and Confers Resistance of Triple-Negative Breast Cancer to Radiotherapy., Liu, Wenjing, Zheng, Min, Zhang, Rou, Jiang, Qiuyun, Du, Guangshi, Wu, Yingying, Yang, Chuanyu, Li, Fubing, Li, Wei, Wang, Luzhen, Wu, Jiao, Shi, Lei, Li, Wenhui, Zhang, Kai, Zhou, Zhongmei, Liu, Rong, Gao, Yingzheng
表观遗传 癌症 调控 代谢 Dna损伤 应答 生物治疗 肿瘤 研究 治疗

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