Professor | 邱菊 | 数字化转型顾问

Name	邱菊
Contact Information	qiuju@sinh.ac.cn
Language ability	中文, 英语
Province	上海

Name

邱菊

Contact Information

qiuju@sinh.ac.cn

Language ability

中文, 英语

Province

上海

University/Lab Affiliation and Title	中国科学院大学
Faculty	Medical School
Academic Background & Achievements	2004-09--2009-04 博士: 上海交通大学医学院 1999-09--2004-07 学士: 泰山医学院
Work Experience	2014-03~现在 - 中国科学院上海生命科学研究院健康科学研究所 - 研究员 2009-11~2014-02 - 美国西北大学费恩伯格医学院 - 博士后 2009-04~2009-10 - 爱因斯坦医学院 - 博士后

University/Lab Affiliation and Title

中国科学院大学

Faculty

Medical School

Academic Background & Achievements

2004-09--2009-04 博士: 上海交通大学医学院
1999-09--2004-07 学士: 泰山医学院

Work Experience

2014-03~现在 - 中国科学院上海生命科学研究院健康科学研究所 - 研究员
2009-11~2014-02 - 美国西北大学费恩伯格医学院 - 博士后
2009-04~2009-10 - 爱因斯坦医学院 - 博士后

Areas of Focus	粘膜免疫肠道免疫天然淋巴细胞
Publications	HTR2A激动剂通过限制ILC2活化在木瓜蛋白酶诱导的肺部炎症中发挥治疗作用。, Wang, Zhishuo, Yan, Chenghua, Du, Qizhen, Huang, Yuying, Li, Xuezhen, Zeng, Dan, Mao, Ruizhi, Gurram, Rama Krishna, Cheng, Shipeng, Gu, Wangpeng, Zhu, Lin, Fan, Weiguo, Ma, Liyan, Ling, Zhiyang, Qiu, Ju, Li, Dangsheng, Liu, Enmei, Zhang, Yaguang, Fang, Yiru, Zhu, Jinfang, Sun, Bing., 2023 Setd2支持GATA3(+)ST2(+)胸腺来源的Treg细胞并抑制肠道炎症。, Ding, Zhaoyun, Cai, Ting, Tang, Jupei, Sun, Hanxiao, Qi, Xinyi, Zhang, Yunpeng, Ji, Yan, Yuan, Liyun, Chang, Huidan, Ma, Yanhui, Zhou, Hong, Li, Li, Sheng, Huiming, Qiu, Ju., 2022 Setd2决定肠道ILC3亚群的不同特性以调节肠道免疫。, Jiali Chang, Xiaojuan Ji, Tian Deng, Jinxin Qiu, Zhaoyun Ding, Zhao Li, Yanhui Ma, Xiaoyu Hu, Li Li, Ju Qiu., 2022 Brg1限制ILC3的促炎特性并调节肠道免疫。, Qi, Xinyi, Qiu, Jinxin, Chang, Jiali, Ji, Yan, Yang, Qi, Cui, Guoliang, Sun, Liming, Chai, Qian, Qin, Jun, Qiu, Ju., 2021 组织信号在ILC2中印记Aiolos表达以调节2型免疫。, Qiu, Jinxin, Zhang, Jingjing, Ji, Yan, Sun, Hanxiao, Gu, Zhitao, Sun, Qiangling, Bai, Meizhu, Gong, Jue, Tang, Jupei, Zhang, Yunpeng, Li, Shiyang, Shao, Zhen, Li, Jinsong, Sheng, Huiming, Shen, Lei, Qiu, Ju., 2021 ILC3来源的OX40L对于免疫缺陷小鼠肠道Tregs的稳态至关重要。, Deng, Tian, Suo, Caixia, Chang, Jiali, Yang, Rui, Li, Jingyu, Cai, Ting, Qiu, Ju., 2020 3型先天淋巴细胞通过LT-LT beta R途径在李斯特菌感染期间指导杯状细胞分化。, Pian, Yaya, Chai, Qian, Ren, Boyang, Wang, Yue, Lv, Mengjie, Qiu, Ju, Zhu, Mingzhao., 2020 ILC3来源的OX40L对于免疫缺陷小鼠肠道Tregs的稳态至关重要。, Tian Deng, Caixia Suo, Jiali Chang, Rui Yang, Jingyu Li, Ting Cai, Ju Qiu., 2019 IL-17产生的ST2(+) 2型先天淋巴细胞在肺部炎症中起致病作用。, Cai, Ting, Qiu, Jinxin, Ji, Yan, Li, Wenjing, Ding, Zhaoyun, Suo, Caixia, Chang, Jiali, Wang, Jingjing, He, Rui, Qian, Youcun, Guo, Xiaohuan, Zhou, Liang, Sheng, Huiming, Shen, Lei, Qiu, Ju., 2019 DR3信号的激活导致ILC3的丧失并加剧肠道炎症。, Jingyu Li, Wenli Shi, Hanxiao Sun, Yan Ji, Yuqin Chen, Xiaohuan Guo, Huiming Sheng, Jie Shu, Liang Zhou, Ting Cai, Ju Qiu., 2019 IL-17产生的ST2+ 2型先天淋巴细胞在肺部炎症中起致病作用。, Ting Cai BS, Jinxin Qiu BS, Yan Ji PhD, Wenjing Li MS, Zhaoyun Ding BS, Caixia Suo PhD,

Areas of Focus

粘膜免疫
肠道免疫
天然淋巴细胞

Publications

HTR2A激动剂通过限制ILC2活化在木瓜蛋白酶诱导的肺部炎症中发挥治疗作用。, Wang, Zhishuo, Yan, Chenghua, Du, Qizhen, Huang, Yuying, Li, Xuezhen, Zeng, Dan, Mao, Ruizhi, Gurram, Rama Krishna, Cheng, Shipeng, Gu, Wangpeng, Zhu, Lin, Fan, Weiguo, Ma, Liyan, Ling, Zhiyang, Qiu, Ju, Li, Dangsheng, Liu, Enmei, Zhang, Yaguang, Fang, Yiru, Zhu, Jinfang, Sun, Bing., 2023
Setd2支持GATA3(+)ST2(+)胸腺来源的Treg细胞并抑制肠道炎症。, Ding, Zhaoyun, Cai, Ting, Tang, Jupei, Sun, Hanxiao, Qi, Xinyi, Zhang, Yunpeng, Ji, Yan, Yuan, Liyun, Chang, Huidan, Ma, Yanhui, Zhou, Hong, Li, Li, Sheng, Huiming, Qiu, Ju., 2022
Setd2决定肠道ILC3亚群的不同特性以调节肠道免疫。, Jiali Chang, Xiaojuan Ji, Tian Deng, Jinxin Qiu, Zhaoyun Ding, Zhao Li, Yanhui Ma, Xiaoyu Hu, Li Li, Ju Qiu., 2022
Brg1限制ILC3的促炎特性并调节肠道免疫。, Qi, Xinyi, Qiu, Jinxin, Chang, Jiali, Ji, Yan, Yang, Qi, Cui, Guoliang, Sun, Liming, Chai, Qian, Qin, Jun, Qiu, Ju., 2021
组织信号在ILC2中印记Aiolos表达以调节2型免疫。, Qiu, Jinxin, Zhang, Jingjing, Ji, Yan, Sun, Hanxiao, Gu, Zhitao, Sun, Qiangling, Bai, Meizhu, Gong, Jue, Tang, Jupei, Zhang, Yunpeng, Li, Shiyang, Shao, Zhen, Li, Jinsong, Sheng, Huiming, Shen, Lei, Qiu, Ju., 2021
ILC3来源的OX40L对于免疫缺陷小鼠肠道Tregs的稳态至关重要。, Deng, Tian, Suo, Caixia, Chang, Jiali, Yang, Rui, Li, Jingyu, Cai, Ting, Qiu, Ju., 2020
3型先天淋巴细胞通过LT-LT beta R途径在李斯特菌感染期间指导杯状细胞分化。, Pian, Yaya, Chai, Qian, Ren, Boyang, Wang, Yue, Lv, Mengjie, Qiu, Ju, Zhu, Mingzhao., 2020
ILC3来源的OX40L对于免疫缺陷小鼠肠道Tregs的稳态至关重要。, Tian Deng, Caixia Suo, Jiali Chang, Rui Yang, Jingyu Li, Ting Cai, Ju Qiu., 2019
IL-17产生的ST2(+) 2型先天淋巴细胞在肺部炎症中起致病作用。, Cai, Ting, Qiu, Jinxin, Ji, Yan, Li, Wenjing, Ding, Zhaoyun, Suo, Caixia, Chang, Jiali, Wang, Jingjing, He, Rui, Qian, Youcun, Guo, Xiaohuan, Zhou, Liang, Sheng, Huiming, Shen, Lei, Qiu, Ju., 2019
DR3信号的激活导致ILC3的丧失并加剧肠道炎症。, Jingyu Li, Wenli Shi, Hanxiao Sun, Yan Ji, Yuqin Chen, Xiaohuan Guo, Huiming Sheng, Jie Shu, Liang Zhou, Ting Cai, Ju Qiu., 2019
IL-17产生的ST2+ 2型先天淋巴细胞在肺部炎症中起致病作用。, Ting Cai BS, Jinxin Qiu BS, Yan Ji PhD, Wenjing Li MS, Zhaoyun Ding BS, Caixia Suo PhD,

粘膜免疫肠道免疫天然淋巴细胞免疫调节炎症 T细胞细胞因子免疫治疗肠道微生物群免疫稳态

周旭宇 - 中国科学院大学

刘丰亮 - 中国科学院大学

Area of Focus
病毒 | 免疫学 | 疫苗 | 病原体 | 免疫反应 | 感染 | 抗体 | T细胞 | B细胞 | 细胞因子

王红艳 - 中国科学院大学

Area of Focus
巨噬细胞 | T细胞 | 炎症 | 疾病 | 调控 | 机制 | 免疫 | 细胞生物学 | 研究 | 创新

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