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毛开睿
maok@xmu.edu.cn
英语, 中文
福建
厦门大学
Life Sciences
  • 2004年 - 生物技术学士学位:山东师范大学
  • 2011年 - 细胞生物学博士学位:中国科学院上海生命科学研究院生化细胞所
  • 2011-2013年 - 研究助理:中国科学院上海生命科学研究院生化细胞所
  • 2013-2019年 - 访问学者:美国国立卫生研究院(NIH)过敏和感染性疾病研究所
  • 2019年至今 - 黏膜免疫课题组组长:厦门大学生命科学学院
肠道菌群与免疫系统的相互作用
  • Thinking differently about ILCs-Not just tissue resident and not just the same as CD4(+) T-cell effectors, Huang, Y.#*, Mao, K.#*, and Germain, R. N., 2018
  • Innate and adaptive lymphocytes sequentially shape the gut microbiota and lipid metabolism, Mao, K.*, Baptista, A. P., Tamoutounour, S., Zhuang, L., Bouladoux, N., Martins, A. J., Huang, Y., Gerner, M. Y., Belkaid, Y., and Germain, R. N.*, 2018
  • S1P-dependent interorgan trafficking of group 2 innate lymphoid cells supports host defense, Huang, Y.*#, Mao, K. #, Chen, X., Sun, M. A., Kawabe, T., Li, W., Usher, N., Zhu, J., Urban, J. F., Jr., Paul, W. E., and Germain, R. N.*, 2018
  • Nitric oxide suppresses NLRP3 inflammasome activation and protects against LPS-induced septic shock, Mao, K. #, Chen, S. #, Chen, M., Ma, Y., Wang, Y., Huang, B., He, Z., Zeng, Y., Hu, Y., Sun, S., Li, J., Wu, X., Wang, X., Strober, W., Chen, C.*, Meng, G.*, and Sun, B*., 2013
  • beta-arrestin1 is critical for the full activation of NLRP3 and NLRC4 inflammasomes, Mao, K. #, Chen, S. #, Wang, Y. #, Zeng, Y., Ma, Y., Hu, Y., Zhang, H., Sun, S., Wu, X., Meng, G., Pei, G.*, and Sun, B*., 2015
  • Group 3 innate lymphoid cells continuously require the transcription factor GATA-3 after commitment, Zhong, C., Cui, K., Wilhelm, C., Hu, G., Mao, K., Belkaid, Y., Zhao, K., and Zhu, J*., 2016
  • Cellular localization of NLRP3 inflammasome, Wang, Y., Yang, C., Mao, K., Chen, S., Meng, G.*, and Sun, B*., 2013
  • Tripartite-motif protein 30 negatively regulates NLRP3 inflammasome activation by modulating reactive oxygen species production, Hu, Y. #, Mao, K. #, Zeng, Y., Chen, S., Tao, Z., Yang, C., Sun, S., Wu, X., Meng, G., and Sun, B*., 2010
  • Trichosanthin functions as Th2-type adjuvant in induction of allergic airway inflammation, Wang, Y., Mao, K., Sun, S., Lin, G., Wu, X., Yao, G., and Sun, B*., 2009
  • TRIM30 alpha negatively regulates TLR-mediated NF-kappa B activation by targeting TAB2 and TAB3 for degradation, Shi, M. #, Deng, W. #, Bi, E., Mao, K., Ji, Y., Lin, G., Wu, X., Tao, Z., Li, Z., Cai, X., Sun, S., Xiang, C.*, and Sun, B*., 2008
肠道菌群 免疫系统 宿主生理 病理 神经系统 免疫功能 肿瘤发生 癌症治疗 组织稳态 基因组技术

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